Related references
Note: Only part of the references are listed.Effects of copper deficiency on mouse yolk sac vasculature and expression of angiogenic mediators
Soo Jin Yang et al.
BIRTH DEFECTS RESEARCH PART B-DEVELOPMENTAL AND REPRODUCTIVE TOXICOLOGY (2006)
Ethanol-induced fetal dysmorphogenesis in the mouse is diminished by high antioxidative capacity of the mother
Parri Wentzel et al.
TOXICOLOGICAL SCIENCES (2006)
Reduced SOD activity and increased neural tube defects in embryos of the sensitive but not of the resistant cohen diabetic rats cultured under diabetic conditions
S Weksler-Zangen et al.
BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY (2003)
Increased superoxide and vascular dysfunction in CuZnSOD-deficient mice
SP Didion et al.
CIRCULATION RESEARCH (2002)
8-Iso-PGF2α administration generates dysmorphogenesis and increased lipid peroxidation in rat embryos in vitro
P Wentzel et al.
TERATOLOGY (2002)
Congenital malformations among infants whose mothers had gestational diabetes or preexisting diabetes
A Åberg et al.
EARLY HUMAN DEVELOPMENT (2001)
Increase of the isoprostane 8-isoprostaglandin F2α in maternal and fetal blood of rats with streptozotocin-induced diabetes:: Evidence of lipid peroxidation
RT Gerber et al.
AMERICAN JOURNAL OF OBSTETRICS AND GYNECOLOGY (2000)
Radicals and oxidative stress in diabetes
IC West
DIABETIC MEDICINE (2000)
Increased mRNA levels of Mn-SOD and catalase in embryos of diabetic rats from a malformation-resistant strain
J Cederberg et al.
DIABETES (2000)
Share Paper
