Journal
TEXAS HEART INSTITUTE JOURNAL
Volume 41, Issue 5, Pages 461-468Publisher
TEXAS HEART INST
DOI: 10.14503/THIJ-13-3466
Keywords
Atrial fibrillation/diagnosis/etiology/physiopathology/prevention & control; biological markers/blood; C-reactive protein/analysis/metabolism; disease management; heart conduction system/physiopathology; inflammation/complications/diagnosis/etiology; myocardial revascularization/adverse effects; polymorphism, genetic; primary prevention; risk factors
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Funding
- Catholic University of Rome [7021526]
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Atrial fibrillation is associated with substantial morbidity and mortality rates. The incompletely understood pathogenesis of this cardiac dysrhythmia makes it difficult to improve approaches to primary and secondary prevention. Evidence has accumulated in regard to a relationship between inflammation and atrial fibrillation. Investigators have correlated the dysrhythmia with myocarditis, pericardiotomy, and C-reactive protein levels, suggesting that inflammation causes atrial fibrillation or participates in its onset and continuation. Conversely, other investigators suggest that atrial fibrillation induces an inflammatory response. In this review, we summarize and critically discuss the nature and clinical role of inflammation and C-reactive protein in atrial fibrillation.
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