Journal
SHOCK
Volume 42, Issue 1, Pages 3-10Publisher
LIPPINCOTT WILLIAMS & WILKINS
DOI: 10.1097/SHK.0000000000000167
Keywords
Alveolar macrophages; trauma; inflammation; blunt chest trauma; ischemia; reperfusion; hemorrhagic shock; burn
Funding
- Deutsche Forschungsgemeinschaft (DFG) [KFO 200, KN 475/5-2, DFG KFO 200, HU 823/3-2]
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Acute lung injury (ALI) and its more severe form, acute respiratory distress syndrome (ARDS), can result from both direct and indirect pulmonary damage caused by trauma and shock. In the course of ALI/ARDS, mediators released from resident cells, such as alveolar macrophages, may act as chemoattractants for invading cells and stimulate local cells to build up a proinflammatory micromilieu. Depending on the trauma setting, the role of alveolar macrophages is differentially defined. This review focuses on alveolar macrophage function after blunt chest trauma, ischemia/reperfusion, hemorrhagic shock, and thermal burns.
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