Related references
Note: Only part of the references are listed.Extracellular heat shock proteins, cellular export vesicles, and the Stress Observation System: A form of communication during injury, infection, and cell damage
Antonio De Maio
CELL STRESS & CHAPERONES (2011)
Trauma Hemorrhagic Shock-Induced Lung Injury Involves a Gut-Lymph-Induced TLR4 Pathway in Mice
Diego C. Reino et al.
PLOS ONE (2011)
PROTEOMIC ANALYSIS OF HUMAN MESENTERIC LYMPH
Monika Dzieciatkowska et al.
SHOCK (2011)
ANTICOAGULANTS INFLUENCE THE IN VITRO ACTIVITY AND COMPOSITION OF SHOCK LYMPH BUT NOT ITS IN VIVO ACTIVITY
Edwin A. Deitch et al.
SHOCK (2011)
Intravenous Injection of Mesenteric Lymph Produced During Hemorrhagic Shock Decreases RBC Deformability in the Rat
Michael Condon et al.
JOURNAL OF TRAUMA-INJURY INFECTION AND CRITICAL CARE (2011)
Neutrophils in the activation and regulation of innate and adaptive immunity
Alberto Mantovani et al.
NATURE REVIEWS IMMUNOLOGY (2011)
A new feature of the stress response: increase in endocytosis mediated by Hsp70
Virginia L. Vega et al.
CELL STRESS & CHAPERONES (2010)
CHANGES IN THE MESENTERIC LYMPH PROTEOME INDUCED BY HEMORRHAGIC SHOCK
Anubhav Mittal et al.
SHOCK (2010)
PROTEOMIC ANALYSIS OF POST-HEMORRHAGIC SHOCK MESENTERIC LYMPH
Jen-Feng Fang et al.
SHOCK (2010)
Estrogenic Hormone Modulation Abrogates Changes in Red Blood Cell Deformability and Neutrophil Activation in Trauma Hemorrhagic Shock
Danielle R. Doucet et al.
JOURNAL OF TRAUMA-INJURY INFECTION AND CRITICAL CARE (2010)
TOLL-LIKE RECEPTOR 4 REGULATES HEME OXYGENASE-1 EXPRESSION AFTER HEMORRHAGIC SHOCK INDUCED ACUTE LUNG INJURY IN MICE: REQUIREMENT OF P38 MITOGEN-ACTIVATED PROTEIN KINASE ACTIVATION
Chang Chen et al.
SHOCK (2009)
HMGB1 IS MARKEDLY ELEVATED WITHIN 6 HOURS OF MECHANICAL TRAUMA IN HUMANS
Erik D. Peltz et al.
SHOCK (2009)
Proteome and system ontology of hemorrhagic shock: Exploring early constitutive changes in postshock mesenteric lymph
Erik D. Peltz et al.
SURGERY (2009)
Anti-inflammatory Resuscitation Improves Survival in Hemorrhage With Trauma
Bolin Cai et al.
JOURNAL OF TRAUMA-INJURY INFECTION AND CRITICAL CARE (2009)
Innate immune adaptor MyD88 mediates neutrophil recruitment and myocardial injury after ischemia-reperfusion in mice
Yan Feng et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2008)
Identification of oxidative stress and toll-like receptor 4 signaling as a key pathway of acute lung injury
Yumiko Imai et al.
CELL (2008)
Systemic inflammation and end organ damage following trauma involves functional TLR4 signaling in both bone marrow-derived cells and parenchymal cells
Kevin P. Mollen et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2008)
Use of Ly6G-specific monoclonal antibody to deplete neutrophils in mice
Jean M. Daley et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2008)
How dying cells alert the immune system to danger
Hajime Kono et al.
NATURE REVIEWS IMMUNOLOGY (2008)
Intravenous injection of trauma-hemorrhagic shock mesenteric lymph causes lung injury that is dependent upon activation of the inducible nitric oxide synthase pathway
Maheswari Senthil et al.
ANNALS OF SURGERY (2007)
Pancreatic duct ligation abrogates the trauma hemorrhage-induced gut barrier failure and the subsequent production of biologically active intestinal lymph
Francis J. Caputo et al.
SHOCK (2007)
The gut is a motor of organ system dysfunction
Cynthia L. Leaphart et al.
SURGERY (2007)
DAMPs, PAMPs and alarmins: all we need to know about danger
Marco E. Bianchi
JOURNAL OF LEUKOCYTE BIOLOGY (2007)
Emerging paradigm: Toll-like receptor 4 - Sentinel for the detection of tissue damage
Kevin P. Mollen et al.
SHOCK (2006)
Systemic inflammation and remote organ damage following bilateral femur fracture requires Toll-like receptor 4
Ryan M. Levy et al.
AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY (2006)
Gut-lymph hypothesis of systemic inflammatory response syndrome/multiple-organ dysfunction syndrome: validating studies in a porcine model
Maheswari Senthil et al.
JOURNAL OF TRAUMA-INJURY INFECTION AND CRITICAL CARE (2006)
Lymph from a primate baboon trauma hemorrhagic shock model activates human neutrophils
EA Deitch et al.
SHOCK (2006)
Toll-like receptor-4 signaling mediates hepatic injury and systemic inflammation in hemorrhagic shock
JM Prince et al.
JOURNAL OF THE AMERICAN COLLEGE OF SURGEONS (2006)
Trauma-hemorrhagic shock mesenteric lymph from rat contains a modified form of albumin that is implicated in endothelial cell toxicity
VL Kaiser et al.
SHOCK (2005)
The role of lymph factors in lung injury, bone marrow suppression, and endothelial cell dysfunction in a primate model of trauma-hemorrhagic shock
EA Deitch et al.
SHOCK (2004)
A study of the biologic activity of trauma-hemorrhagic shock mesenteric lymph over time and the relative role of cytokines
MT Davidson et al.
SURGERY (2004)
Factors in intestinal lymph after shock increase neutrophil adhesion molecule expression and pulmonary leukosequestration
JM Caruso et al.
JOURNAL OF TRAUMA-INJURY INFECTION AND CRITICAL CARE (2003)
Injury primes the innate immune system for enhanced toll-like receptor reactivity
HM Paterson et al.
JOURNAL OF IMMUNOLOGY (2003)
Neutrophils and acute lung injury
E Abraham
CRITICAL CARE MEDICINE (2003)
Ethyl pyruvate prevents lethality in mice with established lethal sepsis and systemic inflammation
L Ulloa et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)
Shock mesenteric lymph-induced rat polymorphonuclear neutrophil activation and endothelial cell injury is mediated by aqueous factors
SD Dayal et al.
JOURNAL OF TRAUMA-INJURY INFECTION AND CRITICAL CARE (2002)
Regulation of Toll-like receptor 4 expression in the lung following hemorrhagic shock and lipopolysaccharide
J Fan et al.
JOURNAL OF IMMUNOLOGY (2002)
The danger model: A renewed sense of self
P Matzinger
SCIENCE (2002)
HMGB1 as a late mediator of lethal systemic inflammation
HH Wang et al.
AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE (2001)
Factors larger than 100 kd in post-hemorrhagic shock mesenteric lymph are toxic for endothelial cells
CA Adams et al.
SURGERY (2001)
The lipid fraction of post-hemorrhagic shock mesenteric lymph (PHSML) inhibits neutrophil apoptosis and enhances cytotoxic potential
RJ Gonzalez et al.
SHOCK (2000)
Share Paper
