Related references
Note: Only part of the references are listed.Lack of reactive oxygen species breaks T cell tolerance to collagen type II and allows development of arthritis in mice
Malin Hultqvist et al.
JOURNAL OF IMMUNOLOGY (2007)
Cutaneous and other lupus-like symptoms in carriers of X-linked chronic granulomatous disease: incidence and autoimmune serology
C. M. Cale et al.
CLINICAL AND EXPERIMENTAL IMMUNOLOGY (2007)
In the absence of reactive oxygen species, T cells default to a Th1 phenotype and mediate protection against pulmonary Cryptococcus neoformans infection
Robert J. Snelgrove et al.
JOURNAL OF IMMUNOLOGY (2006)
Inhibition of human neutrophil IL-8 production by hydrogen peroxide and dysregulation in chronic granulomatous disease
JA Lekstrom-Himes et al.
JOURNAL OF IMMUNOLOGY (2005)
Gene expression profiling provides insight into the pathophysiology of chronic granulomatous disease
SD Kobayashi et al.
JOURNAL OF IMMUNOLOGY (2004)
Deficiency of NADPH oxidase components p47phox and gp91 phox caused granulomatous synovitis and increased connective tissue destruction in experimental arthritis models
FAJ van de Loo et al.
AMERICAN JOURNAL OF PATHOLOGY (2003)
Chemoattractant-stimulated Rac activation in wild-type and Rac2-deficient murine neutrophils: Preferential activation of Rac2 and Rac2 gene dosage effect on neutrophil functions
SJ Li et al.
JOURNAL OF IMMUNOLOGY (2002)
Role of NADPH oxidase in the mechanism of lung neutrophil sequestration and microvessel injury induced by gram-negative sepsis:: Studies in p47phox-/- and gp91phox-/- mice
XP Gao et al.
JOURNAL OF IMMUNOLOGY (2002)
Superoxide prevents nitric oxide-mediated suppression of helper T lymphocytes: Decreased autoimmune encephalomyelitis in nicotinamide adenine dinucleotide phosphate oxidase knockout mice
RC van der Veen et al.
JOURNAL OF IMMUNOLOGY (2000)