Journal
SEMINARS IN CANCER BIOLOGY
Volume 58, Issue -, Pages 109-117Publisher
ACADEMIC PRESS LTD- ELSEVIER SCIENCE LTD
DOI: 10.1016/j.semcancer.2018.08.010
Keywords
Preferential killing; Oxidative stress; Redox homeostasis; Function; Anticancer drugs
Categories
Funding
- Ministry of Science and Technology [MOST 107-2320-B-037-016, MOST 107-2314-B-037-048, MOST 107-2314-B-037-057, MOST 107-2311-B-214-003]
- National Sun Yat-sen University-KMU Joint Research Project [NSYSUKMU 107-P001]
- Kaohsiung Medical University Hospital [KMUH106-6T12]
- Health and Welfare Surcharge of Tobacco Products, the Ministry of Health and Welfare, Taiwan, Republic of China [MOHW107-TDU-B-212-114016]
- Kaohsiung Medical University Research Center [KMU-TC108A04]
Ask authors/readers for more resources
To achieve preferential effects against cancer cells but less damage to normal cells is one of the main challenges of cancer research. In this review, we explore the roles and relationships of oxidative stress-mediated apoptosis, DNA damage, ER stress, autophagy, metabolism, and migration of ROS-modulating anticancer drugs. Understanding preferential anticancer effects in more detail will improve chemotherapeutic approaches that are based on ROS-modulating drugs in cancer treatments.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available