Related references
Note: Only part of the references are listed.Mechanisms That Can Promote Peripheral B-cell Lymphoma in ATM-Deficient Mice
Suprawee Tepsuporn et al.
CANCER IMMUNOLOGY RESEARCH (2014)
Mechanisms of Programmed DNA Lesions and Genomic Instability in the Immune System
Frederick W. Alt et al.
CELL (2013)
The appropriateness of the mouse model for ataxia-telangiectasia: Neurological defects but no neurodegeneration
Martin F. Lavin
DNA REPAIR (2013)
Breakpoint sites disclose the role of the V(D)J recombination machinery in the formation of T-cell receptor (TCR) and non-TCR associated aberrations in T-cell acute lymphoblastic leukemia
Nicole S. D. Larmonie et al.
HAEMATOLOGICA (2013)
The ATM protein kinase: regulating the cellular response to genotoxic stress, and more
Yosef Shiloh et al.
NATURE REVIEWS MOLECULAR CELL BIOLOGY (2013)
The Response to and Repair of RAG-Mediated DNA Double-Strand Breaks
Beth A. Helmink et al.
ANNUAL REVIEW OF IMMUNOLOGY, VOL 30 (2012)
Chromosome Translocation, B Cell Lymphoma, and Activation-Induced Cytidine Deaminase
Davide F. Robbiani et al.
Annual Review of Pathology-Mechanisms of Disease (2012)
Spatial Organization of the Mouse Genome and Its Role in Recurrent Chromosomal Translocations
Yu Zhang et al.
CELL (2012)
Telomere dysfunction and chromosome instability
John P. Murnane
MUTATION RESEARCH-FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS (2012)
Mechanisms that Promote and Suppress Chromosomal Translocations in Lymphocytes
Monica Gostissa et al.
ANNUAL REVIEW OF IMMUNOLOGY, VOL 29 (2011)
The BCL11B tumor suppressor is mutated across the major molecular subtypes of T-cell acute lymphoblastic leukemia
Alejandro Gutierrez et al.
BLOOD (2011)
Translocation-Capture Sequencing Reveals the Extent and Nature of Chromosomal Rearrangements in B Lymphocytes
Isaac A. Klein et al.
CELL (2011)
Genome-wide Translocation Sequencing Reveals Mechanisms of Chromosome Breaks and Rearrangements in B Cells
Roberto Chiarle et al.
CELL (2011)
Understanding the limitations of radiation-induced cell cycle checkpoints
Dorothee Deckbar et al.
CRITICAL REVIEWS IN BIOCHEMISTRY AND MOLECULAR BIOLOGY (2011)
The Mechanism of Double-Strand DNA Break Repair by the Nonhomologous DNA End-Joining Pathway
Michael R. Lieber
ANNUAL REVIEW OF BIOCHEMISTRY, VOL 79 (2010)
Origin of Chromosomal Translocations in Lymphoid Cancer
Andre Nussenzweig et al.
CELL (2010)
ATM-deficient thymic lymphoma is associated with aberrant tcrd rearrangement and gene amplification
Shan Zha et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2010)
Chimeric IgH-TCR/translocations in T lymphocytes mediated by RAG
Elsa Callen et al.
CELL CYCLE (2009)
Long-range oncogenic activation of Igh-c-myc translocations by the Igh 3′ regulatory region
Monica Gostissa et al.
NATURE (2009)
Mechanisms promoting translocations in editing and switching peripheral B cells
Jing H. Wang et al.
NATURE (2009)
Oncogenic transformation in the absence of Xrcc4 targets peripheral B cells that have undergone editing and switching
Jing H. Wang et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2008)
Complementary functions of ATM and H2AX in development and suppression of genomic instability
Shan Zha et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)
ATM prevents the persistence and propagation of chromosome breaks in lymphocytes
Elsa Callen et al.
CELL (2007)
The impact of translocations and gene fusions on cancer causation
Felix Mitelman et al.
NATURE REVIEWS CANCER (2007)
Pathways that suppress programmed DNA breaks from progressing to chromosomal breaks and translocations
Sonia Franco et al.
DNA REPAIR (2006)
Myc translocations in B cell and plasma cell neoplasms
Siegfried Janz
DNA REPAIR (2006)
ATM stabilizes DNA double-strand-break complexes during V(D)J recombination
Andrea L. Bredemeyer et al.
NATURE (2006)
Role of genomic instability and p53 in AID-induced c-myc-Igh translocations
AR Ramiro et al.
NATURE (2006)
H2AX prevents DNA breaks from progressing to chromosome breaks and translocations
S Franco et al.
MOLECULAR CELL (2006)
Evidence for replicative repair of DNA double-strand breaks leading to oncogenic translocation and gene amplification
MJ Difilippantonio et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2002)
Unrepaired DNA breaks in p53-deficient cells lead to oncogenic gene amplification subsequent to translocations
CM Zhu et al.
CELL (2002)
Mechanisms of chromosomal translocations in B cell lymphomas
R Küppers et al.
ONCOGENE (2001)
Interplay of p53 and DNA-repair protein XRCC4 in tumorigenesis, genomic stability and development
YJ Gao et al.
NATURE (2000)
DNA repair protein Ku80 suppresses chromosomal aberrations and malignant transformation
MJ Difilippantonio et al.
NATURE (2000)