Related references
Note: Only part of the references are listed.p73 regulates maintenance of neural stem cell
Massimiliano Agostini et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2010)
The miR-34 family in cancer and apoptosis
H. Hermeking
CELL DEATH AND DIFFERENTIATION (2010)
p73 is an essential regulator of neural stem cell maintenance in embryonal and adult CNS neurogenesis
F. Talos et al.
CELL DEATH AND DIFFERENTIATION (2010)
TAp73 Acts via the bHLH Hey2 to Promote Long-Term Maintenance of Neural Precursors
Masashi Fujitani et al.
CURRENT BIOLOGY (2010)
Isoform-specific p73 knockout mice reveal a novel role for ΔNp73 in the DNA damage response pathway
Margareta T. Wilhelm et al.
GENES & DEVELOPMENT (2010)
p73 deficiency results in impaired self renewal and premature neuronal differentiation of mouse neural progenitors independently of p53
L. Gonzalez-Cano et al.
CELL DEATH & DISEASE (2010)
miR-34a, a microRNA up-regulated in a double transgenic mouse model of Alzheimer's disease, inhibits bcl2 translation
Xiaoying Wang et al.
BRAIN RESEARCH BULLETIN (2009)
Mef2-mediated transcription of the miR379-410 cluster regulates activity-dependent dendritogenesis by fine-tuning Pumilio2 protein levels
Roberto Fiore et al.
EMBO JOURNAL (2009)
Association study between P53 and P73 gene polymorphisms and the sporadic late-onset form of Alzheimer's disease
Renato Scacchi et al.
JOURNAL OF NEURAL TRANSMISSION (2009)
p63 Antagonizes p53 to Promote the Survival of Embryonic Neural Precursor Cells
Chandrasagar B. Dugani et al.
JOURNAL OF NEUROSCIENCE (2009)
Syntaxin 1A is required for normal in utero development
John E. McRory et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2008)
TAp73 knockout shows genomic instability with infertility and tumor suppressor functions
Richard Tomasini et al.
GENES & DEVELOPMENT (2008)
The MYCN oncogene is a direct target of miR-34a
J. S. Wei et al.
ONCOGENE (2008)
TAp73 is a downstream target of p53 in controlling the cellular defense against stress
Jianli Wang et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
Tumor-suppressive miR-34a induces senescence-like growth arrest through modulation of the E2F pathway in human colon cancer cells
Hiroshi Tazawa et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2007)
p53-mediated activation of miRNA34 candidate tumor-suppressor genes
Guido T. Bommer et al.
CURRENT BIOLOGY (2007)
Synaptotagmin I and II are present in distinct subsets of central synapses
Michael A. Fox et al.
JOURNAL OF COMPARATIVE NEUROLOGY (2007)
MicroRNA-34a functions as a potential tumor suppressor by inducing apoptosis in neuroblastoma cells
C. Welch et al.
ONCOGENE (2007)
A microRNA component of the p53 tumour suppressor network
Lin He et al.
NATURE (2007)
Transactivation of miR-34a by p53 broadly influences gene expression and promotes apoptosis
Tsung-Cheng Chang et al.
MOLECULAR CELL (2007)
Transcriptional activation of miR-34a contributes to p53-mediated apoptosis
Nina Raver-Shapira et al.
MOLECULAR CELL (2007)
TAp73α induces tau phosphorylation in HEK293a cells via a transcription-dependent mechanism
Claudie Hooper et al.
NEUROSCIENCE LETTERS (2006)
TAp73/ΔNp73 influences apoptotic response, chemosensitivity and prognosis in hepatocellular carcinoma
M Müller et al.
CELL DEATH AND DIFFERENTIATION (2005)
Roles for p53 and p73 during oligodendrocyte development
N Billon et al.
DEVELOPMENT (2004)
The p53 homologue p73 accumulates in the nucleus and localizes to neurites and neurofibrillary tangles in Alzheimer disease brain
C Wilson et al.
NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY (2004)
Human ΔNp73 regulates a dominant negative feedback loop for TAp73 and p53
TJ Grob et al.
CELL DEATH AND DIFFERENTIATION (2001)
Genome-wide gene expression profiles of the developing mouse hippocampus
M Mody et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2001)
Induction of neuronal differentiation by p73 in a neuroblastoma cell line
V De Laurenzi et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
p73-deficient mice have neurological, pheromonal and inflammatory defects but lack spontaneous tumours
A Yang et al.
NATURE (2000)
Share Paper
