Related references
Note: Only part of the references are listed.Persistence of High-Grade Cervical Dysplasia and Cervical Cancer Requires the Continuous Expression of the Human Papillomavirus Type 16 E7 Oncogene
Sean F. Jabbar et al.
CANCER RESEARCH (2009)
Mutations and Response to Epidermal Growth Factor Receptor Inhibitors
Pierre Laurent-Puig et al.
CLINICAL CANCER RESEARCH (2009)
Bipartite functions of the CREB co-activators selectively direct alternative splicing or transcriptional activation
Antonio L. Amelio et al.
EMBO JOURNAL (2009)
Robust production and passaging of infectious HPV in squamous epithelium of primary human keratinocytes
Hsu-Kun Wang et al.
GENES & DEVELOPMENT (2009)
RNA splicing factors regulated by HPV16 during cervical tumour progression
Sarah Mole et al.
JOURNAL OF PATHOLOGY (2009)
Control of the Papillomavirus Early-to-Late Switch by Differentially Expressed SRp20
Rong Jia et al.
JOURNAL OF VIROLOGY (2009)
Human papillomavirus type 16 E2 and E6 are RNA-binding proteins and inhibit in vitro splicing of pre-mRNAs with suboptimal splice sites
Sohrab Bodaghi et al.
VIROLOGY (2009)
The emerging role of splicing factors in cancer
Ana Rita Grosso et al.
EMBO REPORTS (2008)
HPV-16 RNA processing
Stefan Schwartz
FRONTIERS IN BIOSCIENCE-LANDMARK (2008)
The human keratins: biology and pathology
Roland Moll et al.
HISTOCHEMISTRY AND CELL BIOLOGY (2008)
Binding of DAZAP1 and hnRNPA1/A2 to an exonic splicing silencer in a natural BRCA1 Exon 18 mutant
Elisa Goina et al.
MOLECULAR AND CELLULAR BIOLOGY (2008)
The alternative splicing factor hnRNP A1 is up-regulated during virus-infected epithelial cell differentiation and binds the human papillomavirus type 16 late regulatory element
Thanaporn Cheunim et al.
VIRUS RESEARCH (2008)
Integration of the HPV16 genome does not invariably result in high levels of viral oncogene transcripts
N. Hafner et al.
ONCOGENE (2008)
The large and small isoforms of human papillomavirus type 16 E6 bind to and differentially affect procaspase 8 stability and activity
Maria Filippova et al.
JOURNAL OF VIROLOGY (2007)
The RNA-binding protein Sam68 modulates the alternative splicing of Bcl-x
Maria Paola Paronetto et al.
JOURNAL OF CELL BIOLOGY (2007)
Signals, pathways and splicing regulation
Matias Blaustein et al.
INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY (2007)
Interaction of papillomavirus E2 protein with the Brm chromatin remodeling complex leads to enhanced transcriptional activation
R. Ajay Kumar et al.
JOURNAL OF VIROLOGY (2007)
IKKα controls p52/RelB at the skp2 gene promoter to regulate G1- to S-phase progression
Gueter Schneider et al.
EMBO JOURNAL (2006)
The E7 oncoprotein is translated from spliced E6*I transcripts in high-risk human papillomavirus type 16-or type 18-positive cervical cancer cell lines via translation reinitiation
S Tang et al.
JOURNAL OF VIROLOGY (2006)
The human SWI/SNF subunit Brm is a regulator of alternative splicing
E Batsche et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2006)
Concerted regulation of nuclear and cytoplasmic activities of SR proteins by AKT
M Blaustein et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2005)
Loss of net as repressor leads to constitutive increased c-fos transcription in cervical cancer cells
J van Riggelen et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Signal-dependent regulation of splicing via phosphorylation of Sam68
N Matter et al.
NATURE (2002)
Transcriptional activators differ in their abilities to control alternative splicing
G Nogués et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Papillomaviruses and cancer: From basic studies to clinical application
H zur Hausen
NATURE REVIEWS CANCER (2002)
Leaky scanning is the predominant mechanism for translation of human papillomavirus type 16 E7 oncoprotein from E6/E7 bicistronic mRNA
SN Stacey et al.
JOURNAL OF VIROLOGY (2000)
UV-induced stabilization of c-fos and other short-lived mRNAs
C Blattner et al.
MOLECULAR AND CELLULAR BIOLOGY (2000)
Inhibition of p38 MAP kinase as a therapeutic strategy
JC Lee et al.
IMMUNOPHARMACOLOGY (2000)
The human papillomavirus type 16 E5 protein modulates ERK1/2 and p38 MAP kinase activation by an EGFR-Independent process in stressed human keratinocytes
K Crusius et al.
VIRUS GENES (2000)