Related references
Note: Only part of the references are listed.Copy number, linkage disequilibrium and disease association in the FCGR locus
Heather A. Niederer et al.
HUMAN MOLECULAR GENETICS (2010)
A defunctioning polymorphism in FCGR2B is associated with protection against malaria but susceptibility to systemic lupus erythematosus
Lisa C. Willcocks et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2010)
Negative feedback control of the autoimmune response through antigen-induced differentiation of IL-10-secreting Th1 cells
Leona Gabrysova et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2009)
IP-10-Mediated T Cell Homing Promotes Cerebral Inflammation over Splenic Immunity to Malaria Infection
Catherine Q. Nie et al.
PLOS PATHOGENS (2009)
Immunity to malaria: more questions than answers
Jean Langhorne et al.
NATURE IMMUNOLOGY (2008)
Chemokine receptor CXCR3 and its ligands CXCL9 and CXCL10 are required for the development of murine cerebral malaria
Gabriele S. V. Campanella et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease
Martin S. Weber et al.
NATURE MEDICINE (2007)
Murine cerebral malaria development is independent of Toll-like receptor signaling
Dieudonnee Togbe et al.
AMERICAN JOURNAL OF PATHOLOGY (2007)
Systemic lupus erythematosus-associated defects in the inhibitory receptor FcγRIIb reduce susceptibility to malaria
Menna R. Clatworthy et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2007)
Autoreactive B cell responses to RNA-related antigens due to TLR7 gene duplication
Prapaporn Pisitkun et al.
SCIENCE (2006)
Systemic lupus erythematosus: genes versus environment in high risk populations
M. Molokhia et al.
LUPUS (2006)
Loss of function of a lupus-associated FcγRIIb polymorphism through exclusion from lipid rafts
RA Floto et al.
NATURE MEDICINE (2005)
How malaria has affected the human genome and what human genetics can teach us about malaria
DP Kwiatkowski
AMERICAN JOURNAL OF HUMAN GENETICS (2005)
γδ T cells regulate the extent and duration of inflammation in the central nervous system by a Fas ligand-dependent mechanism
ED Ponomarev et al.
JOURNAL OF IMMUNOLOGY (2005)
Immunological processes in malaria pathogenesis
L Schofield et al.
NATURE REVIEWS IMMUNOLOGY (2005)
Cytokines: accelerators and brakes in the pathogenesis of cerebral malaria
NH Hunt et al.
TRENDS IN IMMUNOLOGY (2003)
Macrophages in chronic type 2 inflammation have a novel phenotype characterized by the abundant expression of Ym1 and Fizz1 that can be partly replicated in vitro
MG Nair et al.
IMMUNOLOGY LETTERS (2003)
Expression of ICOS in vivo defines CD4+ effector T cells with high inflammatory potential and a strong bias for secretion of interleukin 10
M Löhning et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2003)
A novel polymorphism in the Fc gamma receptor IIB (CD32B) transmembrane region alters receptor signaling
XL Li et al.
ARTHRITIS AND RHEUMATISM (2003)
On the pathogenic role of brain-sequestered αβ CD8+ T cells in experimental cerebral malarial
E Belnoue et al.
JOURNAL OF IMMUNOLOGY (2002)
Locally up-regulated lymphotoxin α, not systemic tumor necrosis factor α, is the principle mediator of murine cerebral malaria
CR Engwerda et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2002)
Genetic modifiers of systemic lupus erythematosus in FcγRIIB-/- mice
S Bolland et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2002)