Related references
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The AID antibody diversification enzyme is regulated by protein kinase A phosphorylation
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The mutation spectrum of purified AID is similar to the mutability index in Ramos cells and in ung-/-msh2-/- mice
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Mismatch recognition and uracil excision provide complementary paths to both Ig switching and the A/T-focused phase of somatic mutation
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Processive AID-catalysed cytosine deamination on single-stranded DNA simulates somatic hypermutation
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Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2)
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Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme
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