Related references
Note: Only part of the references are listed.Passive immunotherapy rapidly increases structural plasticity in a mouse model of Alzheimer disease
Tara L. Spires Jones et al.
NEUROBIOLOGY OF DISEASE (2009)
Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory
Ganesh M. Shankar et al.
NATURE MEDICINE (2008)
Plaque-bearing mice with reduced levels of oligomeric amyloid-β assemblies have intact memory function
S. Lesne et al.
NEUROSCIENCE (2008)
Impaired spine stability underlies plaque-related spine loss in an Alzheimer's disease mouse model
Tara L. Spires-Jones et al.
AMERICAN JOURNAL OF PATHOLOGY (2007)
Natural oligomers of the Alzheimer amyloid-β protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway
Ganesh M. Shankar et al.
JOURNAL OF NEUROSCIENCE (2007)
Aβ oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer's disease
Pascale N. Lacor et al.
JOURNAL OF NEUROSCIENCE (2007)
Spatial relationship between synapse loss and β-amyloid deposition in Tg2576 mice
Hongxin Dong et al.
JOURNAL OF COMPARATIVE NEUROLOGY (2007)
A specific amyloid-β protein assembly in the brain impairs memory
S Lesné et al.
NATURE (2006)
Targeting amyloid-β peptide (Aβ) oligomers by passive immunization with a conformation-selective monoclonal antibody improves learning and memory in Aβ precursor protein (APP) transgenic mice
EB Lee et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Dendritic spine abnormalities in amyloid precursor protein transgenic mice demonstrated by gene transfer and intravital multiphoton microscopy
TL Spires et al.
JOURNAL OF NEUROSCIENCE (2005)
Regulation of NMDA receptor trafficking by amyloid-β
EM Snyder et al.
NATURE NEUROSCIENCE (2005)
Natural oligomers of the amyloid-protein specifically disrupt cognitive function
JP Cleary et al.
NATURE NEUROSCIENCE (2005)
Oligomerization of Alzheimer's β-amyloid within processes and synapses of cultured neurons and brain
RH Takahashi et al.
JOURNAL OF NEUROSCIENCE (2004)
Synaptic targeting by Alzheimer's-related amyloid β oligomers
PN Lacor et al.
JOURNAL OF NEUROSCIENCE (2004)
Fibrillar amyloid deposition leads to local synaptic abnormalities and breakage of neuronal branches
J Tsai et al.
NATURE NEUROSCIENCE (2004)
Synaptic slaughter in Alzheimer's disease
PD Coleman et al.
NEUROBIOLOGY OF AGING (2003)
Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo
DM Walsh et al.
NATURE (2002)
Co-expression of multiple transgenes in mouse CNS: a comparison of strategies
JL Jankowsky et al.
BIOMOLECULAR ENGINEERING (2001)