4.8 Article

Loss of MeCP2 in aminergic neurons causes cell-autonomous defects in neurotransmitter synthesis and specific behavioral abnormalities

Related references

Note: Only part of the references are listed.
Article Genetics & Heredity

CSF and urine biogenic amine metabolites in Rett syndrome

Annika Lekman et al.

CLINICAL GENETICS (2010)

Article Biochemistry & Molecular Biology

Mouse models of MeCP2 disorders share gene expression changes in the cerebellum and hypothalamus

Shay Ben-Shachar et al.

HUMAN MOLECULAR GENETICS (2009)

Article Neurosciences

Pathophysiology of Locus Ceruleus Neurons in a Mouse Model of Rett Syndrome

Praveen Taneja et al.

JOURNAL OF NEUROSCIENCE (2009)

Article Neurosciences

Transgenic Mice Lacking Serotonin Neurons Have Severe Apnea and High Mortality during Development

Matthew R. Hodges et al.

JOURNAL OF NEUROSCIENCE (2009)

Article Neurosciences

Non-cell autonomous influence of MeCP2-deficient glia on neuronal dendritic morphology

Nurit Ballas et al.

NATURE NEUROSCIENCE (2009)

Review Medical Laboratory Technology

Clinical utility of monoamine neurotransmitter metabolite analysis in cerebrospinal fluid

Keith Hyland

CLINICAL CHEMISTRY (2008)

Article Biochemistry & Molecular Biology

A partial loss of function allele of Methyl-CpG-binding protein 2 predicts a human neurodevelopmental syndrome

Rodney C. Samaco et al.

HUMAN MOLECULAR GENETICS (2008)

Review Clinical Neurology

The history of dopamine and levodopa in the treatment of Parkinson's disease

Stanley Fahn

MOVEMENT DISORDERS (2008)

Article Multidisciplinary Sciences

MeCP2, a key contributor to neurological disease, activates and represses transcription

Maria Chahrour et al.

SCIENCE (2008)

Article Multidisciplinary Sciences

Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genes

Dag H. Yasui et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2007)

Article Multidisciplinary Sciences

Enhanced anxiety and stress-induced corticosterone release are associated with increased Crh expression in a mouse model of Rett syndrome

Bryan E. McGill et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)

Article Genetics & Heredity

Very mild cases of Rett syndrome with skewed X inactivation

P. Huppke et al.

JOURNAL OF MEDICAL GENETICS (2006)

Article Neurosciences

Mecp2 deficiency disrupts norepinephrine and respiratory systems in mice

JC Viemari et al.

JOURNAL OF NEUROSCIENCE (2005)

Article Multidisciplinary Sciences

A genetic approach to access serotonin neurons for in vivo and in vitro studies

MM Scott et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)

Article Clinical Neurology

Neuropathology of Rett syndrome

DD Armstrong

JOURNAL OF CHILD NEUROLOGY (2005)

Article Biochemistry & Molecular Biology

Abnormalities of social interactions and home-cage behavior in a mouse model of Rett syndrome

P Moretti et al.

HUMAN MOLECULAR GENETICS (2005)

Article Psychology, Biological

Behaviour in the elevated plus-maze predicts coping after subchronic mild stress in mice

C Ducottet et al.

PHYSIOLOGY & BEHAVIOR (2004)

Article Biochemistry & Molecular Biology

X-chromosome inactivation ratios affect wild-type MeCP2 expression within mosaic Rett syndrome and Mecp2-/+ mouse brain

D Braunschweig et al.

HUMAN MOLECULAR GENETICS (2004)

Review Neurosciences

The serotonergic system and anxiety

JA Gordon et al.

NEUROMOLECULAR MEDICINE (2004)

Article Developmental Biology

Transgenic expression of Cre recombinase from the tyrosine hydroxylase locus

J Lindeberg et al.

GENESIS (2004)