Related references
Note: Only part of the references are listed.Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice
Masashi Nakatani et al.
FASEB JOURNAL (2008)
S-nitrosylation of histone deacetylase 2 induces chromatin remodelling in neurons
Alexi Nott et al.
NATURE (2008)
Long-term enhancement of skeletal muscle mass and strength by single gene administration of myostatin inhibitors
Amanda M. Haidet et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)
Nitric oxide release combined with nonsteroidal anti inflammatory activity prevents muscular dystrophy pathology and enhances stem cell therapy
Silvia Brunelli et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2007)
Gene expression profiling in the early phases of DMD: a constant molecular signature characterizes DMD muscle from early postnatal life throughout disease progression
Mario Pescatori et al.
FASEB JOURNAL (2007)
How corticosteroids control inflammation: Quintiles prize lecture 2005
Peter J. Barnes
BRITISH JOURNAL OF PHARMACOLOGY (2006)
Ex vivo treatment with nitric oxide increases mesoangioblast therapeutic efficacy in muscular dystrophy
Clara Sciorati et al.
JOURNAL OF CELL SCIENCE (2006)
Functional and morphological recovery of dystrophic muscles in mice treated with deacetylase inhibitors
G. C. Minetti et al.
NATURE MEDICINE (2006)
A nitric oxide signaling pathway controls CREB-mediated gene expression in neurons
A Riccio et al.
MOLECULAR CELL (2006)
被撤回的出版物: Follistatin induction by nitric oxide through cyclic GMP: a tightly regulated signaling pathway that controls myoblast fusion (Retracted article. See vol. 200, pg. 359, 2013)
A Pisconti et al.
JOURNAL OF CELL BIOLOGY (2006)
Telomerase mediates vascular endothelial growth factor-dependent responsiveness in a rat model of hind limb ischemia
G Zaccagnini et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Deacetylase inhibitors increase muscle cell size by promoting myoblast recruitment and fusion through induction of follistatin
S Iezzi et al.
DEVELOPMENTAL CELL (2004)
Loss of myostatin attenuates severity of muscular dystrophy in mdx mice
KR Wagner et al.
ANNALS OF NEUROLOGY (2002)
Steroids in Duchenne muscular dystrophy: from clinical trials to genomic research
F Muntoni et al.
NEUROMUSCULAR DISORDERS (2002)
Stage-specific modulation of skeletal myogenesis by inhibitors of nuclear deacetylases
S Iezzi et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)
Vascular gene transfer of phosphomimetic endothelial nitric oxide synthase (S1177D) using ultrasound-enhanced destruction of plasmid-loaded microbubbles improves vasoreactivity
C Teupe et al.
CIRCULATION (2002)
A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice
M Wehling et al.
JOURNAL OF CELL BIOLOGY (2001)
Class I histone deacetylases sequentially interact with MyoD and pRb during skeletal myogenesis
PL Puri et al.
MOLECULAR CELL (2001)
A role for histone deacetylase HDAC1 in modulating the transcriptional activity of MyoD: inhibition of the myogenic program
A Mal et al.
EMBO JOURNAL (2001)
DNMT1 binds HDAC2 and a new co-repressor, DMAP1, to form a complex at replication foci
MR Rountree et al.
NATURE GENETICS (2000)
Exit from G1 and S phase of the cell cycle is regulated by repressor complexes containing HDAC-Rb-hSWI/SNF and Rb-hSWI/SNF
HS Zhang et al.
CELL (2000)
Glucocorticoid receptor recruitment of histone deacetylase 2 inhibits interleukin-1 beta-induced histone H4 acetylation on lysines 8 and 12
K Ito et al.
MOLECULAR AND CELLULAR BIOLOGY (2000)