Related references
Note: Only part of the references are listed.Clathrin-dependent entry of severe acute respiratory syndrome coronavirus into target cells expressing ACE2 with the cytoplasmic tail deleted
Yuuki Inoue et al.
JOURNAL OF VIROLOGY (2007)
Cleavage of spike protein of SARS coronavirus by protease factor Xa is associated with viral infectivity
Lanying Du et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2007)
HIV-1 Vpr induces ATM-dependent cellular signal with enhanced homologous recombination
C. Nakai-Murakami et al.
ONCOGENE (2007)
Highly conserved regions within the spike proteins of human coronaviruses 229E and NL63 determine recognition of their respective cellular receptors
Heike Hofmann et al.
JOURNAL OF VIROLOGY (2006)
SARS coronavirus, but not human coronavirus NL63, utilizes cathepsin L to infect ACE2-expressing cells
IC Huang et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
HIV-1 Vpr induces DNA double-strand breaks
H Tachiwana et al.
CANCER RESEARCH (2006)
Human coronavirus NL63, a new respiratory virus
Lia van der Hoek et al.
FEMS MICROBIOLOGY REVIEWS (2006)
Tumor necrosis factor-α convertase (ADAM17) mediates regulated ectodomain shedding of the severe-acute respiratory syndrome-coronavirus (SARS-CoV) receptor, angiotensin-converting enzyme-2 (ACE2)
DW Lambert et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Inhibitors of cathepsin L prevent severe acute respiratory syndrome coronavirus entry
G Simmons et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
A crucial role of angiotensin converting enzyme 2 (ACE2) in SARS coronavirus-induced lung injury
K Kuba et al.
NATURE MEDICINE (2005)
Angiotensin-converting enzyme 2 protects from severe acute lung failure
Y Imai et al.
NATURE (2005)
Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry
H Hofmann et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
Identification of a new human coronavirus
L van der Hoek et al.
NATURE MEDICINE (2004)
Characterization of severe acute respiratory syndrome-associated coronavirus (SARS-CoV) spike glycoprotein-mediated viral entry
G Simmons et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2004)
Interaction between heptad repeat 1 and 2 regions in spike protein of SARS-associated coronavirus: implications for virus fusogenic mechanism and identification of fusion inhibitors
SW Liu et al.
LANCET (2004)
A 193-amino acid fragment of the SARS coronavirus S protein efficiently binds angiotensin-converting enzyme 2
SK Wong et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
Susceptibility to SARS coronavirus S protein-driven infection correlates with expression of angiotensin converting enzyme 2 and infection can be blocked by soluble receptor
H Hofmann et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2004)
pH-dependent entry of Severe acute respiratory syndrome coronavirus is mediated by the spike glycoprotein and enhanced by dendritic cell transfer through DC-SIGN
ZY Yang et al.
JOURNAL OF VIROLOGY (2004)
The spike protein of severe acute respiratory syndrome (SARS) is cleaved in virus infected Vero-E6 cells
XD Wu et al.
CELL RESEARCH (2004)
Angiotensin-converting enzyme 2 is a functional receptor for the SARS coronavirus
WH Li et al.
NATURE (2003)
The ADAMs family of metalloproteases: multidomain proteins with multiple functions
DF Seals et al.
GENES & DEVELOPMENT (2003)
A human homolog of angiotensin-converting enzyme - Cloning and functional expression as a captopril-insensitive carboxypeptidase
SR Tipnis et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
Functional analysis of the domain structure of tumor necrosis factor-α converting enzyme
P Reddy et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
Share Paper
