Related references
Note: Only part of the references are listed.Effect of CCL2 antisense oligodeoxynucleotides on bacterial translocation and subsequent sepsis in severely burned mice orally infected with Enterococcus faecalis
Kenji Shigematsu et al.
EUROPEAN JOURNAL OF IMMUNOLOGY (2012)
Myeloid-Derived Suppressor Cells Evolve during Sepsis and Can Enhance or Attenuate the Systemic Inflammatory Response
Laura Brudecki et al.
INFECTION AND IMMUNITY (2012)
Lack of Th17 Cell Generation in Patients with Severe Burn Injuries
Akihito Inatsu et al.
JOURNAL OF IMMUNOLOGY (2011)
M2b Monocytes Predominated in Peripheral Blood of Severely Burned Patients
Makiko Kobayashi et al.
JOURNAL OF IMMUNOLOGY (2010)
Glycyrrhizin restores the impaired production of beta-defensins in tissues surrounding the burn area and improves the resistance of burn mice to Pseudomonas aeruginosa wound infection
Tsuyoshi Yoshida et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2010)
Enterococcus faecalis translocation in mice with severe burn injury: a pathogenic role of CCL2 and alternatively activated macrophages (M2aMφ and M2cMφ)
Kenji Shigematsu et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2009)
Blood monocytes: distinct subsets, how they relate to dendritic cells, and their possible roles in the regulation of T-cell responses
Frederic Geissmann et al.
IMMUNOLOGY AND CELL BIOLOGY (2008)
A quantitative comparison of rates of phagocytosis and digestion of apoptotic cells by macrophages from normal (BALB/c) and diabetes-prone (NOD) mice
Athanasius F. M. Maree et al.
JOURNAL OF APPLIED PHYSIOLOGY (2008)
Role of polymorphonuclear neutrophils on infectious complications stemming from Enterococcus faecalis oral infection in thermally injured mice
Yasuhiro Tsuda et al.
JOURNAL OF IMMUNOLOGY (2008)
Gr-1+ CD11b+ cells as an accelerator of sepsis stemming from Pseudomonas aeruginosa wound infection in thermally injured mice
Makiko Kobayashi et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2008)
IL-4 and IL-13 negatively regulate TNF-α- and IFN-γ-induced β-defensin expression through STAT-6, suppressor of cytokine signaling (SOCS)-1, and SOCS-3
Cristina Albanesi et al.
JOURNAL OF IMMUNOLOGY (2007)
Mechanism of HBD-3 deficiency in atopic dermatitis
Michael D. Howell et al.
CLINICAL IMMUNOLOGY (2006)
Role of immature myeloid cells in mechanisms of immune evasion in cancer
S Kusmartsev et al.
CANCER IMMUNOLOGY IMMUNOTHERAPY (2006)
Inflammation-associated immune suppression in cancer: The roles played by cytokines, chemokines and additional mediators
A Ben-Baruch
SEMINARS IN CANCER BIOLOGY (2006)
Chronic inflammation, immunosuppression and cancer: New insights and outlook
M Baniyash
SEMINARS IN CANCER BIOLOGY (2006)
Interleukin-10 downregulates anti-microbial peptide expression in atopic dermatitis
MD Howell et al.
JOURNAL OF INVESTIGATIVE DERMATOLOGY (2005)
Beta defensin-3 engineered epidermis shows highly protective effect for bacterial infection
D Sawamura et al.
GENE THERAPY (2005)
Three different neutrophil subsets exhibited in mice with different susceptibilities to infection by methicillin-resistant Staphylococcus aureus
Y Tsuda et al.
IMMUNITY (2004)
Cytokine milieu of atopic dermatitis, as compared to psoriasis, skin prevents induction of innate immune response genes
I Nomura et al.
JOURNAL OF IMMUNOLOGY (2003)
Phagocytosis of apototic cells by macrophages from NOD mice is reduced
BA O'Brien et al.
DIABETES (2002)
β-defensin 1 contributes to pulmonary innate immunity in mice
C Moser et al.
INFECTION AND IMMUNITY (2002)
Innate antimicrobial peptide protects the skin from invasive bacterial infection
V Nizet et al.
NATURE (2001)
Human beta defensin is absent in burn blister fluid
MR Ortega et al.
BURNS (2000)
Share Paper
