Related references
Note: Only part of the references are listed.Assessing the Subcellular Dynamics of Alpha-synuclein Using Photoactivation Microscopy
Susana Goncalves et al.
MOLECULAR NEUROBIOLOGY (2013)
Apolipoprotein E and Alzheimer disease: risk, mechanisms and therapy (vol 9, pg 106, 2013)
Chia-Chen Liu et al.
NATURE REVIEWS NEUROLOGY (2013)
Interaction of α-synuclein with vesicles that mimic mitochondrial membranes
Imola G. Zigoneanu et al.
BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES (2012)
Levels of cerebrospinal fluid neurofilament light protein in healthy elderly vary as a function of TOMM40 variants
Davide Bruno et al.
EXPERIMENTAL GERONTOLOGY (2012)
Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage
Maria Manczak et al.
HUMAN MOLECULAR GENETICS (2011)
Abnormal mitochondrial dynamics, mitochondrial loss and mutant huntingtin oligomers in Huntington's disease: implications for selective neuronal damage
Ulziibat Shirendeb et al.
HUMAN MOLECULAR GENETICS (2011)
Mutant A53T α-Synuclein Induces Neuronal Death by Increasing Mitochondrial Autophagy
Vinay Choubey et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2011)
Dual role of the receptor Tom20 in specificity and efficiency of protein import into mitochondria
Hayashi Yamamoto et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2011)
An Inherited Variable Poly-T Repeat Genotype in TOMM40 in Alzheimer Disease
Allen D. Roses
ARCHIVES OF NEUROLOGY (2010)
Mitochondrial trafficking of APP and alpha synuclein: Relevance to mitochondrial dysfunction in Alzheimer's and Parkinson's diseases
Latha Devi et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2010)
Differential Effects of Wild-Type and A53T Mutant Isoform of Alpha-Synuclein on the Mitochondrial Proteome of Differentiated SH-SY5Y Cells
Kyla Pennington et al.
JOURNAL OF PROTEOME RESEARCH (2010)
Mitochondrial dysfunction in the limelight of Parkinson's disease pathogenesis
Rebecca Banerjee et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2009)
Alpha-synuclein overexpression and aggregation exacerbates impairment of mitochondrial functions by augmenting oxidative stress in human neuroblastoma cells
Mordhwaj S. Parihar et al.
INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY (2009)
Beclin 1 Gene Transfer Activates Autophagy and Ameliorates the Neurodegenerative Pathology in α-Synuclein Models of Parkinson's and Lewy Body Diseases
Brian Spencer et al.
JOURNAL OF NEUROSCIENCE (2009)
Therapeutic approaches to mitochondrial dysfunction in Parkinson's disease
M. Flint Beal
PARKINSONISM & RELATED DISORDERS (2009)
Nature of mitochondrial DNA deletions in substantial nigra neurons
Amy K. Reeve et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2008)
Mitochondrial association of alpha-synuclein causes oxidative stress
M. S. Parihar et al.
CELLULAR AND MOLECULAR LIFE SCIENCES (2008)
Mitochondrial import and accumulation of α-synuclein impair complex I in human dopaminergic neuronal cultures and Parkinson disease brain
Latha Devi et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Proteomics Analysis Identifies Phosphorylation-dependent α-Synuclein Protein Interactions
Melinda A. McFarland et al.
MOLECULAR & CELLULAR PROTEOMICS (2008)
Mitochondria in the aetiology and pathogenesis of Parkinson's disease
Anthony H. V. Schapira
LANCET NEUROLOGY (2008)
Localization of α-synuclein to mitochondria within midbrain of mice
Wen-Wei Li et al.
NEUROREPORT (2007)
ROS, mitochondria and the regulation of autophagy
Ruth Scherz-Shouval et al.
TRENDS IN CELL BIOLOGY (2007)
Accumulation of amyloid precursor protein in the mitochondrial import channels of human Alzheimer's disease brain is associated with mitochondrial dysfunction
Latha Devi et al.
JOURNAL OF NEUROSCIENCE (2006)
High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease
A Bender et al.
NATURE GENETICS (2006)
Mitochondrial DNA deletions are abundant and cause functional impairment in aged human substantia nigra neurons
Y Kraytsberg et al.
NATURE GENETICS (2006)
Mice lacking alpha-synuclein are resistant to mitochondrial toxins
P Klivenyi et al.
NEUROBIOLOGY OF DISEASE (2006)
Parkinson's disease α-synuclein transgenic mice develop neuronal mitochondrial degeneration and cell death
LJ Martin et al.
JOURNAL OF NEUROSCIENCE (2006)
Parkinson-like syndrome induced by continuous MPTP infusion:: Convergent roles of the ubiquitin-proteasome system and α-synuclein
F Fornai et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
Localization of alpha-synuclein to identified fibers and synapses in the normal mouse brain
S Totterdell et al.
NEUROSCIENCE (2005)
Enhanced substantia nigra mitochondrial pathology in human α-synuclein transgenic mice after treatment with MPTP
DD Song et al.
EXPERIMENTAL NEUROLOGY (2004)
α-synuclein locus triplication causes Parkinson's disease
AB Singleton et al.
SCIENCE (2003)
Resistance of α-synuclein null mice to the parkinsonian neurotoxin MPTP
W Dauer et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)
Detection and quantification of mitochondrial DNA deletions in individual cells by real-time PCR
LP He et al.
NUCLEIC ACIDS RESEARCH (2002)
Protein import channel of the outer mitochondrial membrane:: a highly stable Tom40-Tom22 core structure differentially interacts with preproteins, small Tom proteins, and import receptors
C Meisinger et al.
MOLECULAR AND CELLULAR BIOLOGY (2001)
Dopaminergic loss and inclusion body formation in α-synuclein mice:: Implications for neurodegenerative disorders
E Masliah et al.
SCIENCE (2000)
Share Paper
