Journal
PHYTOCHEMISTRY
Volume 70, Issue 13-14, Pages 1571-1580Publisher
PERGAMON-ELSEVIER SCIENCE LTD
DOI: 10.1016/j.phytochem.2009.07.018
Keywords
Wound response; Jasmonate; COI1; JAZ; Systemic signaling; Plant defense
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Funding
- National Institutes of Health [R01GM57795]
- U.S. Department of Energy [DE-FG02-91ER20021]
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Plant tissues are highly vulnerable to injury by herbivores, pathogens, mechanical stress, and other environmental insults. Optimal plant fitness in the face of these threats relies on complex signal transduction networks that link damage-associated signals to appropriate changes in metabolism, growth, and development. Many of these wound-induced adaptive responses are triggered by de novo synthesis of the plant hormone jasmonate (JA). Recent studies provide evidence that JA mediates systemic wound responses through distinct cell autonomous and non-autonomous pathways. In both pathways, bioactive JAs are recognized by an F-box protein-based receptor system that couples hormone binding to ubiquitin-dependent degradation of transcriptional repressor proteins. These results provide a framework for understanding how plants recognize and respond to tissue injury. (c) 2009 Elsevier Ltd. All rights reserved.
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