Related references
Note: Only part of the references are listed.Suppression of Th2 Cytokines Reduces Tick-Transmitted Borrelia burgdorferi Load in Mice
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Cutting edge: CD4 is the receptor for the tick saliva immunosuppressor, Salp15
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A Toll-like receptor 2 ligand stimulates Th2 responses in vivo, via induction of extracellular signal-regulated kinase mitogen-activated protein kinase and c-Fos in dendritic cells
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Glycoprotein 96 can chaperone both MHC class I- and class II-restricted epitopes for in vivo presentation, but selectively primes CD8+ T cell effector function
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Cutting edge: Different toll-like receptor agonists instruct dendritic cells to induce distinct th responses via differential modulation of extracellular signal-regulated kinase-mitogen-activated protein kinase and c-fos
S Agrawal et al.
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CD4 cell priming and tolerization are differentially programmed by APCs upon initial engagement
AD Higgins et al.
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CD8- dendritic cell activation status plays an integral role in influencing Th2 response development
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Identification of an IL-2 binding protein in the saliva of the lyme disease vector tick, Ixodes scapularis
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A filarial nematode-secreted product signals dendritic cells to acquire a phenotype that drives development of Th2 cells
M Whelan et al.
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In vivo CD4+ T cell tolerance induction versus priming is independent of the rate and number of cell divisions
AJ Adler et al.
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