Journal
NUCLEOSIDES NUCLEOTIDES & NUCLEIC ACIDS
Volume 29, Issue 4-6, Pages 453-456Publisher
TAYLOR & FRANCIS INC
DOI: 10.1080/15257771003741380
Keywords
AMP deaminase; heart; C34T mutation; kinetic properties
Categories
Funding
- Ministry of Science of Poland [ST-534, N401 101 31/2201]
Ask authors/readers for more resources
A C34T mutation in the AMPD1 gene is proposed to cause local or systemic augmentations in blood adenosine level and improvement of prognoses in heart diseases like congestive heart failure or heart ischemic disease. This study examines some physico-chemical properties of AMP-deaminase isolated from cardiac muscle of a 10-year-old boy heterozygote for this mutation.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available
Share Paper
