Journal
NITRIC OXIDE-BIOLOGY AND CHEMISTRY
Volume 26, Issue 2, Pages 89-94Publisher
ACADEMIC PRESS INC ELSEVIER SCIENCE
DOI: 10.1016/j.niox.2011.12.005
Keywords
Exercise; Skeletal muscle; Muscle damage; Pain; Hormesis
Categories
Funding
- ETT [38388]
- OTKA [K75702]
- TeT [JAP13/02]
- JSPS [L-10566]
Ask authors/readers for more resources
Skeletal muscle hosts all of the isoforms of nitric oxide synthase (NOS). It is well documented that nitric oxide (NO) regulates force generation and satellite cell activation, and therefore, damage repair of skeletal muscle. NO can also activate nociceptors of C-fibers, thereby causing the sensation of pain. Although delayed-onset of muscle soreness (DOMS) is associated with decreased maximal force generation, pain sensation and sarcomere damage, there is a paucity of research linking NO and DOMS. The present mini-review attempts to elucidate the possible relationship between NO and DOMS, based upon current literature. (c) 2011 Elsevier Inc. All rights reserved.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available