Journal
NEURON
Volume 66, Issue 6, Pages 814-816Publisher
CELL PRESS
DOI: 10.1016/j.neuron.2010.06.014
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Funding
- NINDS NIH HHS [R01 NS020752] Funding Source: Medline
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What happens to a single, presynaptically quiescent synapse among a population of active synapses? In this issue of Neuron, Ehlers and colleagues show that, far from being eliminated, these inactive synapses are primed for potentiation and incorporation into a new neural circuit through an upregulation of NR2B-containing NMDA receptors.
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