Related references
Note: Only part of the references are listed.Sodium Channels in Normal and Pathological Pain
Sulayman D. Dib-Hajj et al.
ANNUAL REVIEW OF NEUROSCIENCE, VOL 33 (2010)
Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel NaV1.7 produce distinct pain disorders
Xiaoyang Cheng et al.
MOLECULAR PAIN (2010)
A Novel Nav1.7 Mutation Producing Carbamazepine-Responsive Erythromelalgia
Tanya Z. Fischer et al.
ANNALS OF NEUROLOGY (2009)
Mexiletine-responsive erythromelalgia due to a new Nav1.7 mutation showing use-dependent current fall-off
Jin-Sung Choi et al.
EXPERIMENTAL NEUROLOGY (2009)
NaV1.7 Gain-of-Function Mutations as a Continuum: A1632E Displays Physiological Changes Associated with Erythromelalgia and Paroxysmal Extreme Pain Disorder Mutations and Produces Symptoms of Both Disorders
M. Estacion et al.
JOURNAL OF NEUROSCIENCE (2008)
Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivation
Brian W. Jarecki et al.
JOURNAL OF PHYSIOLOGY-LONDON (2008)
Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitable
Sulayman D. Dib-Hajj et al.
MOLECULAR PAIN (2008)
Characterization of a new class of potent inhibitors of the voltage-gated sodium channel Nav1.7
Brande S. Williams et al.
BIOCHEMISTRY (2007)
Mutations in sodium-channel gene SCN9A cause a spectrum of human genetic pain disorders
Joost P. H. Drenth et al.
JOURNAL OF CLINICAL INVESTIGATION (2007)
Paroxysmal extreme pain disorder (previously familial rectal pain syndrome)
C. R. Fertleman et al.
NEUROLOGY (2007)
SCN9A mutations in paroxysmal extreme pain disorder:: Allelic variants underlie distinct channel defects and phenotypes
Caroline R. Fertleman et al.
NEURON (2006)
What's in a name - familial rectal pain syndrome becomes paroxysmal extreme pain disorder
C. R. Fertleman et al.
JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY (2006)
A single sodium channel mutation produces hyperor hypoexcitability in different types of neurons
Anthony M. Rush et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)
Sensory and electrophysiological properties of guinea-pig sensory neurones expressing Nav1.7 (PN1) Na+ channel α subunit protein
L Djouhri et al.
JOURNAL OF PHYSIOLOGY-LONDON (2003)
Share Paper
