Journal
NATURE GENETICS
Volume 42, Issue 6, Pages 492-494Publisher
NATURE PUBLISHING GROUP
DOI: 10.1038/ng.585
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Funding
- Kay Kendall Leukemia Fund
- Leukemia Research (UK)
- Cancer Research UK [C1298/A8362]
- Children with Leukemia
- Tumorzentrum Heidelberg-Mannheim, EU [Food-CT-2005-016320, Health-F3-2007-2007-200767]
- Deutsche Krebshilfe
- Hungarian Scientific Research Fund [T042500]
- Economic Competitiveness Operational Programme, Hungary [GVOP 3.1.1-2004-05-0022/3.0]
- NKTH (National Research and Technology) [TECH_ 08-A1/2-2008-0120]
- National Sciences and Engineering Research Council Canada
- NATIONAL CANCER INSTITUTE [R01CA026038] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF GENERAL MEDICAL SCIENCES [T32GM008243] Funding Source: NIH RePORTER
- Medical Research Council [MC_U137686856] Funding Source: researchfish
- MRC [MC_U137686856] Funding Source: UKRI
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Using data from a genome-wide association study of 907 individuals with childhood acute lymphoblastic leukemia (cases) and 2,398 controls and with validation in samples totaling 2,386 cases and 2,419 controls, we have shown that common variation at 9p21.3 (rs3731217, intron 1 of CDKN2A) influences acute lymphoblastic leukemia risk (odds ratio = 0.71, P = 3.01 x 10(-11)), irrespective of cell lineage.
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