Journal
NATURE GENETICS
Volume 41, Issue 8, Pages 866-868Publisher
NATURE PUBLISHING GROUP
DOI: 10.1038/ng0809-866
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Funding
- Medical Research Council [G0700733] Funding Source: Medline
- MRC [G0700733] Funding Source: UKRI
- Medical Research Council [G0300662B, G0700733] Funding Source: researchfish
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A new study reports the first mouse model for ATR-mutated seckel syndrome. The mice show phenotypes recapitulating the human disorder and provide insights into how reduced ATR function affects normal embryonic development by increasing replicative stress, ultimately resulting in an accelerated aging phenotype postnatally.
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