Journal
NATURE CELL BIOLOGY
Volume 12, Issue 2, Pages 104-106Publisher
NATURE PUBLISHING GROUP
DOI: 10.1038/ncb0210-104
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Recent studies have revealed a prominent role of mitochondrial dysfunction in the development of one of the most common neurodegenerative disorders, Parkinson's disease. The ubiquitin ligase Parkin and the protein kinase PINK1, whose mutations are associated with Parkinson's disease, function in a pathway that links ubiquitylation with selective autophagy of damaged mitochondria.
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