Related references
Note: Only part of the references are listed.FADD Deficiency Impairs Early Hematopoiesis in the Bone Marrow
Stephen Rosenberg et al.
JOURNAL OF IMMUNOLOGY (2011)
Fas-associated death domain (FADD) is a negative regulator of T-cell receptor-mediated necroptosis
Stephanie L. Osborn et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2010)
Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced Inflammation
YoungSik Cho et al.
CELL (2009)
Receptor Interacting Protein Kinase-3 Determines Cellular Necrotic Response to TNF-α
Sudan He et al.
CELL (2009)
RIP3, an Energy Metabolism Regulator That Switches TNF-Induced Cell Death from Apoptosis to Necrosis
Duan-Wu Zhang et al.
SCIENCE (2009)
Antigen-mediated T cell expansion regulated by parallel pathways of death
Irene L. Ch'en et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)
The Fas-associated death domain protein is required in apoptosis and TLR-induced proliferative responses in B cells
Hongxia Z. Imtiyaz et al.
JOURNAL OF IMMUNOLOGY (2006)
Conditional fas-associated death domain protein (FADD): GFP knockout mice reveal FADD is dispensable in thymic development but essential in peripheral T cell homeostasis
YH Zhang et al.
JOURNAL OF IMMUNOLOGY (2005)
Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury
A Degterev et al.
NATURE CHEMICAL BIOLOGY (2005)
Essential roles of receptor-interacting protein and TRAF2 in oxidative stress-induced cell death
HM Shen et al.
MOLECULAR AND CELLULAR BIOLOGY (2004)
A role for tumor necrosis factor receptor-2 and receptor-interacting protein in programmed necrosis and antiviral responses
FKM Chan et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
The death domain kinase RIP protects thymocytes from tumor necrosis factor receptor type 2-induced cell death
N Cusson et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2002)
Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule
N Holler et al.
NATURE IMMUNOLOGY (2000)
Share Paper
