Related references
Note: Only part of the references are listed.Frequency of CCR5 genotypes in HIV-infected patients in Roraima, Brazil
Andre de Lima Guerra Corado et al.
BRAZILIAN JOURNAL OF INFECTIOUS DISEASES (2016)
Mitochondria as ATP consumers in cellular pathology
Christos Chinopoulos et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2010)
Diffusion is capable of translating anisotropic apoptosis initiation into a homogeneous execution of cell death
Heinrich J. Huber et al.
BMC SYSTEMS BIOLOGY (2010)
Single-cell quantification of Bax activation and mathematical modelling suggest pore formation on minimal mitochondrial Bax accumulation
H. Duessmann et al.
CELL DEATH AND DIFFERENTIATION (2010)
被撤回的出版物: Activated macrophages utilize glycolytic ATP to maintain mitochondrial membrane potential and prevent apoptotic cell death(Retracted article. See vol. 24, pg. 1132, 2017)
A. Garedew et al.
CELL DEATH AND DIFFERENTIATION (2010)
Systems biology of apoptosis signaling networks
Inna N. Lavrik
CURRENT OPINION IN BIOTECHNOLOGY (2010)
Transient Oxidative Stress Damages Mitochondrial Machinery Inducing Persistent β-Cell Dysfunction
Ning Li et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2009)
Kinetics of Transport and Phosphorylation of Glucose in Cancer Cells
Sara Rodriguez-Enriquez et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2009)
Visualization of ATP levels inside single living cells with fluorescence resonance energy transfer-based genetically encoded indicators
Hiromi Imamura et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2009)
Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation
Matthew G. Vander Heiden et al.
SCIENCE (2009)
GLUT1 and GLUT9 as major contributors to glucose influx in HepG2 cells identified by a high sensitivity intramolecular FRET glucose sensor
Hitomi Takanaga et al.
BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES (2008)
Analysis of cardiac mitochondrial Na+-Ca2+ exchanger kinetics with a biophysical model of mitochondrial Ca2+ handing suggests a 3: 1 stoichiometry
Ranjan K. Dash et al.
JOURNAL OF PHYSIOLOGY-LONDON (2008)
Quantitative analysis of pathways controlling extrinsic apoptosis in single cells
John G. Albeck et al.
MOLECULAR CELL (2008)
BCL-2 family antagonists for cancer therapy
Guillaume Lessene et al.
NATURE REVIEWS DRUG DISCOVERY (2008)
Dynamics of intracellular oxygen in PC12 cells upon stimulation of neurotransmission
Alexander V. Zhdanov et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Universally sloppy parameter sensitivities in systems biology models
Ryan N. Gutenkunst et al.
PLOS COMPUTATIONAL BIOLOGY (2007)
Computer modeling of mitochondrial tricarboxylic acid cycle, oxidative phosphorylation, metabolite transport, and electrophysiology
Fan Wu et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
GAPDH and autophagy preserve survival after apoptotic cytochrome c release in the absence of caspase activation
Anna Colell et al.
CELL (2007)
APOPTO-CELL -: a simulation tool and interactive database for analyzing cellular susceptibility to apoptosis
Heinrich J. Huber et al.
BIOINFORMATICS (2007)
Relationship between Thermodynamic Driving Force and One-Way Fluxes in Reversible Processes
Daniel A. Beard et al.
PLOS ONE (2007)
Systems analysis of effector caspase activation and its control by X-linked inhibitor of apoptosis protein
Markus Rehm et al.
EMBO JOURNAL (2006)
Different mitochondrial-intermembrane space proteins are released during apoptosis in a manner that is coordinately initiated but can vary in duration
Cristina Munoz-Pinedo et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)
OPA1 controls apoptotic cristae remodeling independently from mitochondrial fusion
Christian Frezza et al.
CELL (2006)
A biophysical model of the mitochondrial respiratory system and oxidative phosphorylation
DA Beard
PLOS COMPUTATIONAL BIOLOGY (2005)
Energy substrate modulates mitochondrial structure and oxidative capacity in cancer cells
R Rossignol et al.
CANCER RESEARCH (2004)
Mathematical modeling reveals threshold mechanism in CD95-induced apoptosis
M Bentele et al.
JOURNAL OF CELL BIOLOGY (2004)
The pathophysiology of mitochondrial cell death
DR Green et al.
SCIENCE (2004)
Akt stimulates aerobic glycolysis in cancer cells
RL Elstrom et al.
CANCER RESEARCH (2004)
Real-time single cell analysis of Smac/DIABLO release during apoptosis
M Rehm et al.
JOURNAL OF CELL BIOLOGY (2003)
Training-induced adaptation of oxidative phosphorylation in skeletal muscles
B Korzeniewski et al.
BIOCHEMICAL JOURNAL (2003)
Mitochondrial membrane permeabilization and superoxide production during apoptosis -: A single-cell analysis
H Düssmann et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Regulation of the Apaf-1/Caspase-9 apoptosome by caspase-3 and XIAP
H Zou et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Outer mitochondrial membrane permeabilization during apoptosis triggers caspase-independent mitochondrial and caspase-dependent plasma membrane potential depolarization:: a single-cell analysis
H Düssmann et al.
JOURNAL OF CELL SCIENCE (2003)
Caspase-mediated loss of mitochondrial function and generation of reactive oxygen species during apoptosis
JE Ricci et al.
JOURNAL OF CELL BIOLOGY (2003)
Complex I-mediated reactive oxygen species generation:: modulation by cytochrome c and NAD(P)+ oxidation-reduction state
Y Kushnareva et al.
BIOCHEMICAL JOURNAL (2002)
A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis
L Scorrano et al.
DEVELOPMENTAL CELL (2002)
The mechanism of mitochondrial membrane potential retention following release of cytochrome c in apoptotic GT1-7 neural cells
AC Rego et al.
CELL DEATH AND DIFFERENTIATION (2001)
Cytochrome c maintains mitochondrial transmembrane potential and ATP generation after outer mitochondrial membrane permeabilization during the apoptotic process
NJ Waterhouse et al.
JOURNAL OF CELL BIOLOGY (2001)
Akt and Bcl-xL promote growth factor-independent survival through distinct effects on mitochondrial physiology
DR Plas et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
Mitochondrial precursor signal peptide induces a unique permeability transition and release of cytochrome c from liver and brain mitochondria
YE Kushnareva et al.
ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS (2001)
Cytochrome c is released from mitochondria in a reactive oxygen species (ROS)-dependent fashion and can operate as a ROS scavenger and as a respiratory substrate in cerebellar neurons undergoing excitotoxic death
A Atlante et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis
S Matsuyama et al.
NATURE CELL BIOLOGY (2000)
Mitochondrial membrane potential and neuronal glutamate excitotoxicity: mortality and millivolts
DG Nicholls et al.
TRENDS IN NEUROSCIENCES (2000)
Internally quenched fluorescent peptide substrates disclose the subsite preferences of human caspases 1, 3, 6, 7 and 8
HR Stennicke et al.
BIOCHEMICAL JOURNAL (2000)
The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant
JC Goldstein et al.
NATURE CELL BIOLOGY (2000)
Share Paper
