Journal
MOLECULAR CELL
Volume 52, Issue 5, Pages 611-612Publisher
CELL PRESS
DOI: 10.1016/j.molcel.2013.11.018
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Funding
- Intramural NIH HHS [ZIA BC008710-36] Funding Source: Medline
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In this issue of Molecular Cell, Aarke et al. (2013) identify a toxin-antitoxin system in Caulobacter crescentus that acts by a unique mechanism. The toxin, which blocks DNA replication, is constitutively degraded by CIpXP, and this degradation requires the antitoxin, a CIpXP adaptor.
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