Related references
Note: Only part of the references are listed.The complexity of DNA double strand break is a crucial factor for activating ATR signaling pathway for G2/M checkpoint regulation regardless of ATM function
Lian Xue et al.
DNA REPAIR (2015)
The complexity of DNA double strand breaks is a critical factor enhancing end-resection
Hirohiko Yajima et al.
DNA REPAIR (2013)
Inhibition of ATR kinase with the selective inhibitor VE-821 results in radiosensitization of cells of promyelocytic leukaemia (HL-60)
Jirina Vavrova et al.
RADIATION AND ENVIRONMENTAL BIOPHYSICS (2013)
Targeting radiation-resistant hypoxic tumour cells through ATR inhibition
I. M. Pires et al.
BRITISH JOURNAL OF CANCER (2012)
The novel ATR inhibitor VE-821 increases sensitivity of pancreatic cancer cells to radiation and chemotherapy
Remko Prevo et al.
CANCER BIOLOGY & THERAPY (2012)
Repair of DNA damage induced by accelerated heavy ionsuA mini review
Ryuichi Okayasu
INTERNATIONAL JOURNAL OF CANCER (2012)
Factors determining DNA double-strand break repair pathway choice in G2 phase
Atsushi Shibata et al.
EMBO JOURNAL (2011)
Selective killing of ATM- or p53-deficient cancer cells through inhibition of ATR
Philip M. Reaper et al.
NATURE CHEMICAL BIOLOGY (2011)
Unrepaired clustered DNA lesions induce chromosome breakage in human cells
Aroumougame Asaithamby et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2011)
ATR: a master conductor of cellular responses to DNA replication stress
Rachel Litman Flynn et al.
TRENDS IN BIOCHEMICAL SCIENCES (2011)
Charged particles in radiation oncology
Marco Durante et al.
NATURE REVIEWS CLINICAL ONCOLOGY (2010)
Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexes
L Zou et al.
SCIENCE (2003)
The Human Papillomavirus E6 protein and its contribution to malignant progression
F Mantovani et al.
ONCOGENE (2001)
Share Paper
