Journal
LEARNING & MEMORY
Volume 16, Issue 6, Pages 367-370Publisher
COLD SPRING HARBOR LAB PRESS, PUBLICATIONS DEPT
DOI: 10.1101/lm.1231009
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Funding
- NMH [P50-MH064045]
- NIH [T32NS00743-11]
- NSF Minority Postdoctoral Fellowship [0706858]
- Direct For Biological Sciences
- Div Of Biological Infrastructure [0706858] Funding Source: National Science Foundation
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It is well established that cAMP signaling within neurons plays a major role in the formation of long-term memories-signaling thought to proceed through protein kinase A (PKA). However, here we show that exchange protein activated by cAMP (Epac) is able to enhance the formation of long-term memory in the hippocampus and appears to do so independent of PKA, thus demonstrating the importance of Epac-mediated signaling in memory consolidation.
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