Journal
KIDNEY INTERNATIONAL
Volume 76, Issue 6, Pages 604-613Publisher
ELSEVIER SCIENCE INC
DOI: 10.1038/ki.2009.224
Keywords
apoptosis; cisplatin; cyclin-dependent kinase; necrosis
Categories
Funding
- National Institute of Diabetes and Digestive and Kidney Diseases [R01-DK-54471]
- VA Merit Review
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Acute kidney injury (AKI) activates pathways of cell death and cell proliferation. Although seemingly discrete and unrelated mechanisms, these pathways can now be shown to be connected and even to be controlled by similar pathways. The dependence of the severity of renal-cell injury on cell cycle pathways can be used to control and perhaps to prevent acute kidney injury. This review is written to address the correlation between cellular life and death in kidney tubules, especially in acute kidney injury.
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