Related references
Note: Only part of the references are listed.Maternal Exposure to Dioxin Disrupts Gonadotropin Production in Fetal Rats and Imprints Defects in Sexual Behavior
Tomoki Takeda et al.
JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS (2009)
Dioxin receptor is a ligand-dependent E3 ubiquitin ligase
Fumiaki Ohtake et al.
NATURE (2007)
Suppression of fetal testicular cytochrome P450 17 by maternal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin:: A mechanism involving an initial effect on gonadotropin synthesis in the pituitary
Junko Taketoh et al.
LIFE SCIENCES (2007)
Ontogeny of hepatic CYP1A2 and CYP2E1 expression in rat
Fawzy A. Elbarbry et al.
JOURNAL OF BIOCHEMICAL AND MOLECULAR TOXICOLOGY (2007)
For dioxin-induced birth defects, mouse or human CYP1A2 in maternal liver protects whereas mouse CYP1A1 and CYP1B1 are inconsequential
Nadine Dragin et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Fetal pituitary gonadotropin as an initial target of dioxin in its impairment of cholesterol transportation and steroidogenesis in rats
J Mutoh et al.
ENDOCRINOLOGY (2006)
Effects of acute and chronic exposure to the aryl hydrocarbon receptor agonist 2,3,7,8-tetrachlorodibenzo-p-dioxin on the transition to reproductive senescence in female Sprague-Dawley rats
A Franczak et al.
BIOLOGY OF REPRODUCTION (2006)
Modulation of oestrogen receptor signalling by association with the activated dioxin receptor
F Ohtake et al.
NATURE (2003)
Protection of the Cyp1a2(-/-) null mouse against uroporphyria and hepatic injury following exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin
AG Smith et al.
TOXICOLOGY AND APPLIED PHARMACOLOGY (2001)
Human health effects of dioxins: cancer, reproductive and endocrine system effects
M Kogevinas
HUMAN REPRODUCTION UPDATE (2001)
Maternal exposure to a low dose of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) suppressed the development of reproductive organs of male rats:: Dose-dependent increase of mRNA levels of 5α-reductase type 2 in contrast to decrease of androgen receptor in the pubertal ventral prostate
S Ohsako et al.
TOXICOLOGICAL SCIENCES (2001)
2,3,7,8-Tetraclilovodibenzo-p-dioxin inhibits luminal cell differentiation and androgen responsiveness of the ventral prostate without inhibiting prostatic 5α-dihydrotestosterone formation or testicular androgen production in rat offspring
HM Theobald et al.
TOXICOLOGICAL SCIENCES (2000)
Toxicologic evidence of developmental neurotoxicity of environmental chemicals
HR Andersen et al.
TOXICOLOGY (2000)
Acute administration of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in pregnant Long Evans rats:: Association of measured tissue concentrations with developmental effects
CH Hurst et al.
TOXICOLOGICAL SCIENCES (2000)
Share Paper
