Related references
Note: Only part of the references are listed.Ischaemic and morphine-induced post-conditioning: impact of mKCa channels
R. Huhn et al.
BRITISH JOURNAL OF ANAESTHESIA (2010)
Activation of big conductance Ca2+-activated K+ channels (BK) protects the heart against ischemia-reperfusion injury
Bo Hjorth Bentzen et al.
PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY (2009)
Persistent beneficial effect of postconditioning against infarct size:: role of mitochondrial KATP channels during reperfusion
James Mykytenko et al.
BASIC RESEARCH IN CARDIOLOGY (2008)
Na+/H+ exchanger inhibitor cariporide attenuates the mitochondrial Ca2+ overload and PTP opening
Takako Toda et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)
Interaction of cardiovascular risk factors with myocardial ischemia/reperfusion injury, preconditioning, and postconditioning
Peter Ferdinandy et al.
PHARMACOLOGICAL REVIEWS (2007)
Opening of mitochondrial permeability transition pore induces hypercontracture in Ca2+ overloaded cardiac myocytes
Marisol Ruiz-Meana et al.
BASIC RESEARCH IN CARDIOLOGY (2007)
The mitochondrial Ca2+-activated K+ channel activator, NS 1619 inhibits L-type Ca2+ channels in rat ventricular myocytes
Won Sun Park et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2007)
Postconditioning protects human atrial muscle through the activation of the RISK pathway
Vivek Sivaraman et al.
BASIC RESEARCH IN CARDIOLOGY (2007)
Intermittent activation of bradykinin B2 receptors and mitochondrial KATP channels trigger cardiac postconditioning through redox signaling
Claudia Penna et al.
CARDIOVASCULAR RESEARCH (2007)
Post-conditioning reduces infarct size in the isolated rat heart: Role of coronary flow and pressure and the nitric oxide/cGMP pathway
C Penna et al.
BASIC RESEARCH IN CARDIOLOGY (2006)
Post-conditioning induced cardioprotection requires signaling through a redox-sensitive mechanism, mitochondrial ATP-sensitive K+ channel and protein kinase C activation
C Penna et al.
BASIC RESEARCH IN CARDIOLOGY (2006)
Cardiac mitochondrial preconditioning by Big Ca2+-sensitive K+ channel opening requires superoxide radical generation
DF Stowe et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2006)
Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload
HY Sun et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2005)
Calcium-activated potassium channel triggers cardioprotection of ischemic preconditioning
CM Cao et al.
JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS (2005)
The reperfusion injury salvage kinase pathway: A common target for both ischemic preconditioning and postconditioning
DJ Hausenloy et al.
TRENDS IN CARDIOVASCULAR MEDICINE (2005)
Postconditioning attenuates myocardial ischemia-reperfusion injury by inhibiting events in the early minutes of reperfusion
H Kin et al.
CARDIOVASCULAR RESEARCH (2004)
Mitochondrial permeability transition pore opening during myocardial reperfusion - a target for cardioprotection
AP Halestrap et al.
CARDIOVASCULAR RESEARCH (2004)
Preconditioning protects by inhibiting the mitochondrial permeability transition
DJ Hausenloy et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2004)
Opening of Ca2+-activated K+ channels triggers early and delayed preconditioning against I/R injury independent of NOS in mice
XY Wang et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2004)
Preconditioning the myocardium: From cellular physiology to clinical cardiology
DM Yellon et al.
PHYSIOLOGICAL REVIEWS (2003)
Mitochondrial potassium transport:: the role of the mitochondrial ATP-sensitive K+ channel in cardiac function and cardioprotection
KD Garlid et al.
BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS (2003)
Lysophosphatidylcholine-induced myocardial damage is inhibited by pretreatment with poloxamer 188 in isolated rat heart
M Watanabe et al.
MOLECULAR AND CELLULAR BIOCHEMISTRY (2003)
Mechanistically distinct steps in the mitochondrial death pathway triggered by oxidative stress in cardiac myocytes
M Akao et al.
CIRCULATION RESEARCH (2003)
Cytoprotective role of Ca2+-activated K+ channels in the cardiac inner mitochondrial membrane
WH Xu et al.
SCIENCE (2002)
H2O2-induced Ca2+ overload in NRVM involves ERK1/2 MAP kinases:: role for an NHE-1-dependent pathway
EC Rothstein et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2002)
Increase in cytosolic Ca2+ levels through the activation of non-selective cation channels induced by oxidative stress causes mitochondrial depolarization leading to apoptosis-like death in Leishmania donovani promastigotes
SB Mukherjee et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Consequences of brief ischemia: Stunning, preconditioning, and their clinical implications - Part 2
RA Kloner et al.
CIRCULATION (2001)
Mechanisms of Ca2+ overload induced by extracellular H2O2 in quiescent isolated rat cardiomyocytes
W Gen et al.
BASIC RESEARCH IN CARDIOLOGY (2001)
Acetylcholine, bradykinin, opioids, and phenylephrine, but not adenosine, trigger preconditioning by generating free radicals and opening mitochondrial KATP channels
MV Cohen et al.
CIRCULATION RESEARCH (2001)
Mitochondria and Ca2+ in cell physiology and pathophysiology
MR Duchen
CELL CALCIUM (2000)
Share Paper
