Related references
Note: Only part of the references are listed.A small number of open Ca2+ channels trigger transmitter release at a central GABAergic synapse
Iancu Bucurenciu et al.
NATURE NEUROSCIENCE (2010)
Enhanced Excitatory Transmission at Cortical Synapses as the Basis for Facilitated Spreading Depression in Cav2.1 Knockin Migraine Mice
Angelita Tottene et al.
NEURON (2009)
A limited contribution of Ca2+ current facilitation to paired-pulse facilitation of transmitter release at the rat calyx of Held
Martin Mueller et al.
JOURNAL OF PHYSIOLOGY-LONDON (2008)
The S218L familial hemiplegic migraine mutation promotes deinhibition of Cav2.1 calcium channels during direct G-protein regulation
Norbert Weiss et al.
PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY (2008)
Differential gating and recruitment of P/Q-, N-, and R-Type Ca2+ channels in hippocampal mossy fiber boutons
Liyi Li et al.
JOURNAL OF NEUROSCIENCE (2007)
The brain is hyperexcitable in migraine
S. K. Aurora et al.
CEPHALALGIA (2007)
Changes in synaptic transmission properties due to the expression of N-type calcium channels at the calyx of Held synapse of mice lacking P/Q-type calcium channels
Carlota Gonzalez Inchauspe et al.
JOURNAL OF PHYSIOLOGY-LONDON (2007)
Robust correlations between action potential duration and the properties of synaptic connections in layer 4 interneurones in neocortical slices from juvenile rats and adult rat and cat
Afia B. Ali et al.
JOURNAL OF PHYSIOLOGY-LONDON (2007)
Properties of action-potential initiation in neocortical pyramidal cells: Evidence from whole cell axon recordings
Yousheng Shu et al.
JOURNAL OF NEUROPHYSIOLOGY (2007)
Amplitude and kinetics of action potential-evoked Ca2+ current and its efficacy in triggering transmitter release at the developing calyx of held synapse
Yi-Mei Yang et al.
JOURNAL OF NEUROSCIENCE (2006)
Gating deficiency in a familial hemiplegic migraine type 1 mutant P/Q-type calcium channel
CF Barrett et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Specific kinetic alterations of human CaV2.1 calcium channels produced by mutation S218L causing familial hemiplegic migraine and delayed cerebral edema and coma after minor head trauma
A Tottene et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Dynamic aspects of presynaptic calcium currents mediating synaptic transmission
T Takahashi
CELL CALCIUM (2005)
Developmental transformation of the release modality at the calyx of held synapse
MJ Fedchyshyn et al.
JOURNAL OF NEUROSCIENCE (2005)
Effects of familial hemiplegic migraine type 1 mutations on neuronal P/Q-type Ca2+ channel activity and inhibitory synaptic transmission
YQ Cao et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
Gene dosage-dependent transmitter release changes at neuromuscular synapses of Cacna1a R192Q knockin mice are non-progressive and do not lead to morphological changes or muscle weakness
S Kaja et al.
NEUROSCIENCE (2005)
A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression
AMJM van den Maagdenberg et al.
NEURON (2004)
Functional compensation of P/Q by N-type channels blocks short-term plasticity at the calyx of held presynaptic terminal
CG Inchauspe et al.
JOURNAL OF NEUROSCIENCE (2004)
Probing the intracellular calcium sensitivity of transmitter release during synaptic facilitation
F Felmy et al.
NEURON (2003)
Familial hemiplegic migraine mutations increase Ca2+ influx through single human CaV2.1 channels and decrease maximal CaV2.1 current density in neurons
A Tottene et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)
Three new familial hemiplegic migraine mutants affect P/Q-type Ca2+ channel kinetics
RL Kraus et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)