Related references
Note: Only part of the references are listed.N-cadherin Regulates p38 MAPK Signaling via Association with JNK-associated Leucine Zipper Protein IMPLICATIONS FOR NEURODEGENERATION IN ALZHEIMER DISEASE
Koichi Ando et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2011)
Aberrant Amyloid Precursor Protein (APP) Processing in Hereditary Forms of Alzheimer Disease Caused by APP Familial Alzheimer Disease Mutations Can Be Rescued by Mutations in the APP GxxxG Motif
Lisa-Marie Munter et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2010)
Structure and biochemical analysis of the heparin-induced E1 dimer of the amyloid precursor protein
Sven O. Dahms et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2010)
Substrate docking to γ-secretase allows access of γ-secretase modulators to an allosteric site
Kengo Uemura et al.
NATURE COMMUNICATIONS (2010)
Induced Dimerization of the Amyloid Precursor Protein Leads to Decreased Amyloid-β Protein Production
Simone Eggert et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2009)
N-cadherin-based adhesion enhances A beta release and decreases A beta(42/40) ratio
Kengo Uemura et al.
JOURNAL OF NEUROCHEMISTRY (2009)
Allosteric Modulation of PS1/γ-Secretase Conformation Correlates with Amyloid β42/40 Ratio
Kengo Uemura et al.
PLOS ONE (2009)
Dimerization of the transmembrane domain of amyloid precursor proteins and familial Alzheimer's disease mutants
Paul M. Gorman et al.
BMC NEUROSCIENCE (2008)
Homophilic interactions of the amyloid precursor protein (APP) ectodomain are regulated by the loop region and affect β-secretase cleavage of APP
Daniela Kaden et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Amyloidogenic processing but not amyloid precursor protein (APP) intracellular C-terminal domain production requires a precisely oriented APP dimer assembled by transmembrane GXXXG motifs
Pascal Kienlen-Campard et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Endocytosis is required for synaptic activity-dependent release of amyloid-β in vivo
John R. Cirrito et al.
NEURON (2008)
GxxxG motifs within the amyloid precursor protein transmembrane sequence are critical for the etiology of Aβ42
Lisa-Marie Munter et al.
EMBO JOURNAL (2007)
Structural studies of the Alzheimer's amyloid precursor protein copper-binding domain reveal how it binds copper ions
Geoffrey K. -W. Kong et al.
JOURNAL OF MOLECULAR BIOLOGY (2007)
A highly sensitive protein-protein interaction assay based on Gaussia luciferase
Ingrid Remy et al.
NATURE METHODS (2006)
Characterization of sequential N-cadherin cleavage by ADAM10 and PS1
Kengo Uemura et al.
NEUROSCIENCE LETTERS (2006)
Amyloid precursor protein and BACE function as oligomers
Gerd Multhaup
NEURODEGENERATIVE DISEASES (2006)
The amyloid-β precursor protein:: integrating structure with biological function
C Reinhard et al.
EMBO JOURNAL (2005)
Homo- and heterodimerization of APP family members promotes intercellular adhesion
P Soba et al.
EMBO JOURNAL (2005)
NMDA receptor activation inhibits α-secretase and promotes neuronal amyloid-β production
S Lesné et al.
JOURNAL OF NEUROSCIENCE (2005)
Familial Alzheimer's disease presenilin 1 mutations cause alterations in the conformation of presenilin and interactions with amyloid precursor protein
O Berezovska et al.
JOURNAL OF NEUROSCIENCE (2005)
Human BACE forms dimers and colocalizes with APP
A Schmechel et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
The X-ray structure of an antiparaliel dimer of the human amyloid precursor protein E2 domain
YC Wang et al.
MOLECULAR CELL (2004)
PS1 activates PI3K thus inhibiting GSK-3 activity and tau overphosphorylation: effects of FAD mutations
L Baki et al.
EMBO JOURNAL (2004)
Nonsteroidal anti-inflammatory drugs lower Aβ42 and change presenilin 1 conformation
A Lleó et al.
NATURE MEDICINE (2004)
Exclusively targeting β-secretase to lipid rafts by GPI-anchor addition up-regulates β-site processing of the amyloid precursor protein
JM Cordy et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2003)
Demonstration by FRET of BACE interaction with the amyloid precursor protein at the cell surface and in early endosomes
A Kinoshita et al.
JOURNAL OF CELL SCIENCE (2003)
Structure of the Alzheimer's disease amyloid precursor protein copper binding domain - A regulator of neuronal copper homeostasis
KJ Barnham et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Medicine - The amyloid hypothesis of Alzheimer's disease: Progress and problems on the road to therapeutics
J Hardy et al.
SCIENCE (2002)
Cadherin regulates dendritic spine morphogenesis
H Togashi et al.
NEURON (2002)
A presenilin-1/γ-secretase cleavage releases the E-cadherin intracellular domain and regulates disassembly of adherens junctions
P Marambaud et al.
EMBO JOURNAL (2002)
Homodimerization of amyloid precursor protein and its implication in the amyloidogenic pathway of Alzheimer's disease
S Scheuermann et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
Extracellular domains drive homo- but not heterodimerization of erbB receptors
KM Ferguson et al.
EMBO JOURNAL (2000)
Share Paper
