Journal
JOURNAL OF MOLECULAR MEDICINE-JMM
Volume 91, Issue 8, Pages 907-916Publisher
SPRINGER HEIDELBERG
DOI: 10.1007/s00109-013-1060-y
Keywords
Calcium; Calmodulin; Ca-calmodulin-dependent protein kinase II (CaMKII)
Funding
- NIH [P01-HL080101, R37-HL30077, R01-130933]
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Calcium (Ca2+) has long been recognized as a crucial intracellular messenger attaining stimuli-specific cellular outcomes via localized signaling. Ca2+-binding proteins, such as calmodulin (CaM), and its target proteins are key to the segregation and refinement of these Ca2+-dependent signaling events. This review not only summarizes the recent technological advances enabling the study of subcellular Ca2+-CaM and Ca2+-CaM-dependent protein kinase (CaMKII) signaling events but also highlights the outstanding challenges in the field.
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