Journal
JOURNAL OF INNATE IMMUNITY
Volume 4, Issue 1, Pages 80-85Publisher
KARGER
DOI: 10.1159/000334251
Keywords
Ischemia/reperfusion; Kidney; RAGE; HMGB1
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Funding
- NIGMS NIH HHS [R01 GM062508] Funding Source: Medline
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The receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not affect renal injury and function upon I/R-induced injury. Copyright (C) 2011 S. Karger AG, Basel
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