Related references
Note: Only part of the references are listed.Spatial Organization of the Mouse Genome and Its Role in Recurrent Chromosomal Translocations
Yu Zhang et al.
CELL (2012)
DNA damage defines sites of recurrent chromosomal translocations in B lymphocytes
Ofir Hakim et al.
NATURE (2012)
Double-Strand Break End Resection and Repair Pathway Choice
Lorraine S. Symington et al.
ANNUAL REVIEW OF GENETICS, VOL 45 (2011)
Mechanisms that Promote and Suppress Chromosomal Translocations in Lymphocytes
Monica Gostissa et al.
ANNUAL REVIEW OF IMMUNOLOGY, VOL 29 (2011)
Translocation-Capture Sequencing Reveals the Extent and Nature of Chromosomal Rearrangements in B Lymphocytes
Isaac A. Klein et al.
CELL (2011)
Genome-wide Translocation Sequencing Reveals Mechanisms of Chromosome Breaks and Rearrangements in B Cells
Roberto Chiarle et al.
CELL (2011)
53BP1-mediated DNA double strand break repair: Insert bad pun here
Angela T. Noon et al.
DNA REPAIR (2011)
BLM-DNA2-RPA-MRN and EXO1-BLM-RPA-MRN constitute two DNA end resection machineries for human DNA break repair
Amitabh V. Nimonkar et al.
GENES & DEVELOPMENT (2011)
Regulation of DNA End Joining, Resection, and Immunoglobulin Class Switch Recombination by 53BP1
Anne Bothmer et al.
MOLECULAR CELL (2011)
H2AX prevents CtIP-mediated DNA end resection and aberrant repair in G1-phase lymphocytes
Beth A. Helmink et al.
NATURE (2011)
CtIP promotes microhomology-mediated alternative end joining during class-switch recombination
Mieun Lee-Theilen et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2011)
An essential role for CtIP in chromosomal translocation formation through an alternative end-joining pathway
Yu Zhang et al.
NATURE STRUCTURAL & MOLECULAR BIOLOGY (2011)
Inhibition of helicase activity by a small molecule impairs Werner syndrome helicase (WRN) function in the cellular response to DNA damage or replication stress
Monika Aggarwal et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2011)
Origin of Chromosomal Translocations in Lymphoid Cancer
Andre Nussenzweig et al.
CELL (2010)
53BP1 Inhibits Homologous Recombination in Brca1-Deficient Cells by Blocking Resection of DNA Breaks
Samuel F. Bunting et al.
CELL (2010)
53BP1 regulates DNA resection and the choice between classical and alternative end joining during class switch recombination
Anne Bothmer et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2010)
Phosphorylation of Exo1 modulates homologous recombination repair of DNA double-strand breaks
Emma Bolderson et al.
NUCLEIC ACIDS RESEARCH (2010)
Alternative end-joining catalyzes robust IgH locus deletions and translocations in the combined absence of ligase 4 and Ku70
Cristian Boboila et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2010)
ATM-Dependent and -Independent Dynamics of the Nuclear Phosphoproteome After DNA Damage
Ariel Bensimon et al.
SCIENCE SIGNALING (2010)
Genomic Instability Resulting from Blm Deficiency Compromises Development, Maintenance, and Function of the B Cell Lineage
Holger Babbe et al.
JOURNAL OF IMMUNOLOGY (2009)
CUP Links DNA Double-Strand Break Sensing to Resection
Zhongsheng You et al.
MOLECULAR CELL (2009)
Mechanism and regulation of class switch recombination
Janet Stavnezer et al.
ANNUAL REVIEW OF IMMUNOLOGY (2008)
Sgs1 helicase and two nucleases Dna2 and Exo1 resect DNA double-strand break ends
Zhu Zhu et al.
CELL (2008)
AID Is Required for the Chromosomal Breaks in c-myc that Lead to c-myc/IgH Translocations
Davide F. Robbiani et al.
CELL (2008)
DNA helicases Sgs1 and BLM promote DNA double-strand break resection
Serge Gravel et al.
GENES & DEVELOPMENT (2008)
Sae2, Exo1 and Sgs1 collaborate in DNA double-strand break processing
Eleni P. Mimitou et al.
NATURE (2008)
A forward chemical genetic screen reveals an inhibitor of the Mre11-Rad50-Nbs1 complex
Aude Dupre et al.
NATURE CHEMICAL BIOLOGY (2008)
Human premature aging, DNA repair and RecQ helicases
Robert M. Brosh et al.
NUCLEIC ACIDS RESEARCH (2007)
Human CtIP promotes DNA end resection
Alessandro A. Sartori et al.
NATURE (2007)
ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage
Shuhei Matsuoka et al.
SCIENCE (2007)
The impact of translocations and gene fusions on cancer causation
Felix Mitelman et al.
NATURE REVIEWS CANCER (2007)
Pathways that suppress programmed DNA breaks from progressing to chromosomal breaks and translocations
Sonia Franco et al.
DNA REPAIR (2006)
Mutation of the Murine Bloom's syndrome gene produces global genome destabilization
Nicholas Chester et al.
MOLECULAR AND CELLULAR BIOLOGY (2006)
The role of double-strand break repair - insights from human genetics
M O'Driscoll et al.
NATURE REVIEWS GENETICS (2006)
Inactivation of CtIP leads to early embryonic lethality mediated by G1 restraint and to tumorigenesis haploid insufficiency
PL Chen et al.
MOLECULAR AND CELLULAR BIOLOGY (2005)
Opinion -: Somatic hypermutation at A•T pairs:: polymerase error versus dUTP incorporation
MS Neuberger et al.
NATURE REVIEWS IMMUNOLOGY (2005)
Altered somatic hypermutation and reduced class-switch recombination in exonuclease 1-mutant mice
PD Bardwell et al.
NATURE IMMUNOLOGY (2004)
ATM is required for efficient recombination between immunoglobulin switch regions
B Reina-San-Martin et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2004)
53BP1 is required for class switch recombination
IM Ward et al.
JOURNAL OF CELL BIOLOGY (2004)
53BP1 links DNA damage-response pathways to immunoglobulin heavy chain class-switch recombination
JP Manis et al.
NATURE IMMUNOLOGY (2004)
Inactivation of Exonuclease 1 in mice results in DNA mismatch repair defects, increased cancer susceptibility, and male and female sterility
KC Wei et al.
GENES & DEVELOPMENT (2003)
Genomic instability in mice lacking histone H2AX
A Celeste et al.
SCIENCE (2002)
AID is required to initiate Nbs1/γ-H2AX focus formation and mutations at sites of class switching
S Petersen et al.
NATURE (2001)
Cancer predisposition caused by elevated mitotic recombination in Bloom mice
GB Luo et al.
NATURE GENETICS (2000)
Functional link of BRCA1 and ataxia telangiectasia gene product in DNA damage response
S Li et al.
NATURE (2000)
Nuclear localization and cell cycle-specific expression of CtIP, a protein that associates with the BRCA1 tumor suppressor
X Yu et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme
M Muramatsu et al.
CELL (2000)
Share Paper
