Related references
Note: Only part of the references are listed.DNA-PKcs and Artemis function in the end-joining phase of immunoglobulin heavy chain class switch recombination
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JOURNAL OF EXPERIMENTAL MEDICINE (2008)
Non-homologous end-joining, a sticky affair
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IgH class switching and translocations use a robust non-classical end-joining pathway
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The catalytic subunit of DNA-protein kinase (DNA-PKcs) is not required for Ig class-switch recombination
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Pathways that suppress programmed DNA breaks from progressing to chromosomal breaks and translocations
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H2AX prevents DNA breaks from progressing to chromosome breaks and translocations
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Artemis-independent functions of DNA-dependent protein kinase in Ig heavy chain class switch recombination and development
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Impact of DNA ligase IV on nonhomologous end joining pathways during class switch recombination in human cells
Q Pan-Hammarström et al.
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53BP1 is required for class switch recombination
IM Ward et al.
JOURNAL OF CELL BIOLOGY (2004)
53BP1 links DNA damage-response pathways to immunoglobulin heavy chain class-switch recombination
JP Manis et al.
NATURE IMMUNOLOGY (2004)
Reduced switching in SCID B cells is associated with altered somatic mutation of recombined S regions
AJL Cook et al.
JOURNAL OF IMMUNOLOGY (2003)
H2AX is required for recombination between immunoglobulin switch regions but not for intra-switch region recombination or somatic hypermutation
B Reina-San-Martin et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2003)
DNA-dependent protein kinase activity is not required for immunoglobulin class switching
GC Bosma et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2002)
IgH class switch recombination to IgG1 in DNA-PKcs-deficient B cells
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Mechanism and control of class-switch recombination
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TRENDS IN IMMUNOLOGY (2002)
Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2)
P Revy et al.
CELL (2000)
Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme
M Muramatsu et al.
CELL (2000)