Journal
JOURNAL OF COMPUTER ASSISTED TOMOGRAPHY
Volume 35, Issue 1, Pages 39-42Publisher
LIPPINCOTT WILLIAMS & WILKINS
DOI: 10.1097/RCT.0b013e3181f31917
Keywords
posterior reversible encephalopathy syndrome; cerebral amyloid angiopathy; vasogenic edema
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The case of a 75-year-old man with a history of lymphoma, recent upper respiratory tract infection, and a protracted course of encephalopathy is presented. Radiologically, findings were consistent with posterior reversible encephalopathy syndrome. A brain biopsy revealed evidence of endothelial activation, T-cell trafficking, and vascular endothelial growth factor expression, suggesting that systemic immune system activation may be involved with triggering posterior reversible encephalopathy syndrome. In addition, underlying cerebral amyloid angiopathy may have contributed to the initial nonclassical edema distribution by compromising autoregulatory blood flow mechanisms.
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