Related references
Note: Only part of the references are listed.The Temporal Expression, Cellular Localization, and Inhibition of the Chemokines MIP-2 and MCP-1 after Traumatic Brain Injury in the Rat
Jonathan K. J. Rhodes et al.
JOURNAL OF NEUROTRAUMA (2009)
Mouse closed head injury model induced by a weight-drop device
Michael A. Flierl et al.
NATURE PROTOCOLS (2009)
EFFECTS OF MONOCYTE CHEMOATTRACTANT PROTEIN 1 ON BLOOD-BORNE CELL RECRUITMENT AFTER TRANSIENT FOCAL CEREBRAL ISCHEMIA IN MICE
M. Schilling et al.
NEUROSCIENCE (2009)
Serum IL-8 and MCP-1 Concentration Do Not Identify Patients With Enlarging Contusions After Traumatic Brain Injury
Jonathan Rhodes et al.
JOURNAL OF TRAUMA-INJURY INFECTION AND CRITICAL CARE (2009)
Modulation of immune response by head injury
Maria Cristina Morganti-Kossmann et al.
INJURY-INTERNATIONAL JOURNAL OF THE CARE OF THE INJURED (2007)
Multiplex assessment of cytokine and chemokine levels in cerebrospinal fluid following severe pediatric traumatic brain injury: Effects of moderate hypothermia
Sandra D. W. Buttram et al.
JOURNAL OF NEUROTRAUMA (2007)
Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration
Nicole Bye et al.
EXPERIMENTAL NEUROLOGY (2007)
Monocyte chemoattractant protein-1 regulation of blood-brain barrier permeability
SM Stamatovic et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2005)
Blood monocytes consist of two principal subsets with distinct migratory properties
F Geissmann et al.
IMMUNITY (2003)
MCP-1 (CCL2) protects human neurons and astrocytes from NMDA or HIV-tat-induced apoptosis
EA Eugenin et al.
JOURNAL OF NEUROCHEMISTRY (2003)
Overexpression of monocyte chemoattractant protein 1 in the brain exacerbates ischemic brain injury and is associated with recruitment of inflammatory cells
Y Chen et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2003)
Ablation of the chemokine monocyte chemoattractant protein-1 delays retrograde neuronal degeneration, attenuates microglial activation, and alters expression of cell death molecules
MJ Muessel et al.
MOLECULAR BRAIN RESEARCH (2002)
Monocyte recruitment and myelin removal are delayed following spinal cord injury in mice with CCR2 chemokine receptor deletion
MH Ma et al.
JOURNAL OF NEUROSCIENCE RESEARCH (2002)
CCR2 regulates the level of MCP-1/CCL2 in vitro and at inflammatory sites and controls T cell activation in response to alloantigen
LA Tylaska et al.
CYTOKINE (2002)
Distribution, cellular localization and functional role of CCR2 chemokine receptors in adult rat brain
G Banisadr et al.
JOURNAL OF NEUROCHEMISTRY (2002)
Monocyte chemoattractant protein-1 deficiency is protective in a murine stroke model
PM Hughes et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2002)
Role of cerebral inflammation after traumatic brain injury: A revisited concept
MC Morganti-Kossmann et al.
SHOCK (2001)
Absence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitis
DR Huang et al.
JOURNAL OF EXPERIMENTAL MEDICINE (2001)
Early and specific expression of Monocyte Chemoattractant Protein-1 in the thalamus induced by cortical injury
MJ Muessel et al.
BRAIN RESEARCH (2000)
Experimental closed head injury: Analysis of neurological outcome, blood-brain barrier dysfunction, intracranial neutrophil infiltration, and neuronal cell death in mice deficient in genes for pro-inflammatory cytokines
PF Stahel et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2000)
Microglial and astrocytic reactions prior to onset of thalamic cell death after traumatic lesion of the rat sensorimotor cortex
DM Hermann et al.
ACTA NEUROPATHOLOGICA (2000)
Share Paper
