Related references
Note: Only part of the references are listed.MOG1 A New Susceptibility Gene for Brugada Syndrome
Darouna Kattygnarath et al.
CIRCULATION-CARDIOVASCULAR GENETICS (2011)
Transient outward current (Ito) gain-of-function mutations in the KCND3-encoded Kv4.3 potassium channel and Brugada syndrome
John R. Giudicessi et al.
HEART RHYTHM (2011)
An international compendium of mutations in the SCN5A-encoded cardiac sodium channel in patients referred for Brugada syndrome genetic testing
Jamie D. Kapplinger et al.
HEART RHYTHM (2010)
J wave syndromes
Charles Antzelevitch et al.
HEART RHYTHM (2010)
Gain-of-function mutation S422L in the KCNJ8-encoded cardiac KATP channel Kir6.1 as a pathogenic substrate for J-wave syndromes
Argelia Medeiros-Domingo et al.
HEART RHYTHM (2010)
Mutations in the cardiac L-type calcium channel associated with inherited J-wave syndromes and sudden cardiac death
Elena Burashnikov et al.
HEART RHYTHM (2010)
A transient outward potassium current activator recapitulates the electrocardiographic manifestations of Brugada syndrome
Kirstine Calloe et al.
CARDIOVASCULAR RESEARCH (2009)
SCN5A Mutations and the Role of Genetic Background in the Pathophysiology of Brugada Syndrome
Vincent Probst et al.
CIRCULATION-CARDIOVASCULAR GENETICS (2009)
A Mutation in the β3 Subunit of the Cardiac Sodium Channel Associated With Brugada ECG Phenotype
Dan Hu et al.
CIRCULATION-CARDIOVASCULAR GENETICS (2009)
Empiric quinidine therapy for asymptomatic Brugada syndrome: Time for a prospective registry
Sami Viskin et al.
HEART RHYTHM (2009)
Functional Effects of KCNE3 Mutation and Its Role in the Development of Brugada Syndrome
Eva Delpon et al.
CIRCULATION-ARRHYTHMIA AND ELECTROPHYSIOLOGY (2008)
Sudden cardiac death secondary to antidepressant and antipsychotic drugs
Serge Sicouri et al.
EXPERT OPINION ON DRUG SAFETY (2008)
Mutation in glycerol-3-phosphate dehydrogenase 1-like gene (GPD1-L) decreases cardiac Na+ current and causes inherited Arrhythmias
Barry London et al.
CIRCULATION (2007)
Loss-of-function mutations in the cardiac calcium channel underlie a new clinical entity characterized by ST-Segment elevation, short QT intervals, and sudden cardiac death
Charles Antzelevitch et al.
CIRCULATION (2007)
Brugada electrocardiographic pattern due to tricyclic antidepressant overdose
M Akhtar et al.
JOURNAL OF ELECTROCARDIOLOGY (2006)
Dimethyl lithospermate B, an extract of Danshen, suppresses arrhythmogenesis associated with the Brugada syndrome
JM Fish et al.
CIRCULATION (2006)
Brugada syndrome - Report of the second consensus conference - Endorsed by the Heart Rhythm Society and the European Heart Rhythm Association
C Antzelevitch et al.
CIRCULATION (2005)
Tricyclic antidepressants, QT interval prolongation, and Torsade de Pointes
WVR Vieweg et al.
PSYCHOSOMATICS (2004)
State-dependent block of voltage-gated Na+ channels by amitriptyline via the local anesthetic receptor and its implication for neuropathic pain
GK Wang et al.
PAIN (2004)
Ionic and cellular basis for the predominance of the Brugada syndrome phenotype in males
JM Di Diego et al.
CIRCULATION (2002)
Overdose of cyclic antidepressants and the Brugada syndrome
D Goldgran-Toledano et al.
NEW ENGLAND JOURNAL OF MEDICINE (2002)
Transient ST segment elevation in right precordial leads induced by psychotropic drugs: Relationship to the Brugada syndrome
F Rouleau et al.
JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY (2001)