Related references
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Voltage-gated rearrangements associated with differential β-subunit modulation of the L-type Ca2+ channel inactivation
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A β2 adrenergic receptor signaling complex assembled with the Ca2+ channel Cav1.2
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Functional embryonic cardiomyocytes after disruption of the L-type α1C (Cav1.2) calcium channel gene in the mouse
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Ca2+-sensitive inactivation and facilitation of L-type Ca2+ channels both depend on specific amino acid residues in a consensus calmodulin-binding motif in theα1c subunit
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