Related references
Note: Only part of the references are listed.The sensitivity of pancreatic β-cells to mitochondrial injuries triggered by lipotoxicity and oxidative stress
Ning Li et al.
BIOCHEMICAL SOCIETY TRANSACTIONS (2008)
The role for endoplasmic reticulum stress in diabetes mellitus
Decio L. Eizirik et al.
ENDOCRINE REVIEWS (2008)
Mitochondrial respiration and membrane potential after low-flow ischemia are not affected by ischemic preconditioning
Kerstin Boengler et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2007)
Reactive oxygen species as a signal in glucose-stimulated insulin secretion
Jingbo Pi et al.
DIABETES (2007)
The transcription factor PAX4 acts as a survival gene in INS-1E insulinoma cells
T. Brun et al.
ONCOGENE (2007)
Protective role for nitric oxide during the endoplasmic reticulum stress response in pancreatic β-cells
Kajorn Kitiphongspattana et al.
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2007)
Increasing uncoupling protein-2 in pancreatic beta cells does not alter glucose-induced insulin secretion but decreases production of reactive oxygen species
N. Produit-Zengaffinen et al.
DIABETOLOGIA (2007)
Mitochondrial damages and the regulation of insulin secretion
P. Maechler et al.
BIOCHEMICAL SOCIETY TRANSACTIONS (2006)
Mitochondrial DNA-deletion mutations accumulate intracellularly to detrimental levels in aged human skeletal muscle fibers
Entela Bua et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2006)
Mitochondrial respiratory dysfunction and oxidative stress after chronic malathion exposure
Eduardo H. B. Delgado et al.
NEUROCHEMICAL RESEARCH (2006)
Diabetes-associated mitochondrial DNA mutation A3243G impairs cellular metabolic pathways necessary for beta cell function
P. B. M. de Andrade et al.
DIABETOLOGIA (2006)
Parkinson's disease brain mitochondrial complex I has oxidatively damaged subunits and is functionally impaired and misassembled
Paula M. Keeney et al.
JOURNAL OF NEUROSCIENCE (2006)
Somatic mtDNA mutations cause aging phenotypes without affecting reactive oxygen species production
A Trifunovic et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)
Progressive accumulation of mitochondrial DNA mutations and decline in mitochondrial function lead to β-cell failure
RA Simmons et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Insulin secretion profiles are modified by overexpression of glutamate dehydrogenase in pancreatic islets
S Carobbio et al.
DIABETOLOGIA (2004)
Glucose sensitivity and metabolism-secretion coupling studied during two-year continuous culture in INS-1E insulinoma cells
A Merglen et al.
ENDOCRINOLOGY (2004)
Prevention of mitochondrial oxidative damage as a therapeutic strategy in diabetes
K Green et al.
DIABETES (2004)
Mitochondrial formation of reactive oxygen species
JF Turrens
JOURNAL OF PHYSIOLOGY-LONDON (2003)
Uncoupling protein-2 overexpression inhibits mitochondrial death pathway in cardiomyocytes
Y Teshima et al.
CIRCULATION RESEARCH (2003)
Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma
G Mattiasson et al.
NATURE MEDICINE (2003)
Islet β-cell secretion determines glucagon release from neighbouring α-cells
H Ishihara et al.
NATURE CELL BIOLOGY (2003)
Visualizing superoxide production in normal and diabetic rat islets of Langerhans
VP Bindokas et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Glucose toxicity in β-cells:: Type 2 diabetes, good radicals gone bad, and the glutathione connection
RP Robertson et al.
DIABETES (2003)
Oxidative stress-induced iron signaling is responsible for peroxide-dependent oxidation of dichlorodihydrofluorescein in endothelial cells - Role of transferrin receptor-dependent iron uptake in apoptosis
Y Tampo et al.
CIRCULATION RESEARCH (2003)
Mitochondria as the conductor of metabolic signals for insulin exocytosis in pancreatic β-cells
P Maechler
CELLULAR AND MOLECULAR LIFE SCIENCES (2002)
Oxidative stress and stress-activated signaling pathways: A unifying hypothesis of type 2 diabetes
JL Evans et al.
ENDOCRINE REVIEWS (2002)
Angiotensin II stimulation of NAD(P)H oxidase activity - Upstream mediators
PN Seshiah et al.
CIRCULATION RESEARCH (2002)
Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes
S Oyadomari et al.
JOURNAL OF CLINICAL INVESTIGATION (2002)
H2O2-induced Oradical anion2 production by a non-phagocytic NAD(P)H oxidase causes oxidant injury
WG Li et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
β-cell apoptosis -: Stimuli and signaling
T Mandrup-Poulsen
DIABETES (2001)