Related references
Note: Only part of the references are listed.A destructive interaction mechanism accounts for dominant-negative effects of misfolded mutants of voltage-gated calcium channels
Alexandre Mezghrani et al.
JOURNAL OF NEUROSCIENCE (2008)
Dominant-negative effects of episodic ataxia type 2 mutations involve disruption of membrane trafficking of human P/Q-type Ca2+ channels
Chung-Jiuan Jeng et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2008)
Primary episodic ataxias: diagnosis, pathogenesis and treatment
J. C. Jen et al.
BRAIN (2007)
Dominant-negative effects of human P/Q-type Ca2+ channel mutations associated with episodic ataxia type 2
CJ Jeng et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2006)
G protein-gated inhibitory module of N-type (CaV2.2) Ca2+ channels
HL Agler et al.
NEURON (2005)
Dominant-negative calcium channel suppression by truncated constructs involves a kinase implicated in the unfolded protein response
KM Page et al.
JOURNAL OF NEUROSCIENCE (2004)
Translational control in the endoplasmic reticulum stress response
D Ron
JOURNAL OF CLINICAL INVESTIGATION (2002)
Calcium channels and channelopathies of the central nervous system
D Pietrobon
MOLECULAR NEUROBIOLOGY (2002)
Functional consequences of P/Q-type Ca2+ channel Ca(v)2.1 missense mutations associated with episodic ataxia type 2 and progressive ataxia
E Wappl et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Evidence for two concentration-dependent processes for β-subunit effects on α1B calcium channels
C Cantí et al.
BIOPHYSICAL JOURNAL (2001)
The endoplasmic reticulum: integration of protein folding, quality control, signaling and degradation
E Chevet et al.
CURRENT OPINION IN STRUCTURAL BIOLOGY (2001)
Molecular basis for KATP assembly:: Transmembrane interactions mediate association of a K+ channel with an ABC transporter
B Schwappach et al.
NEURON (2000)
Share Paper
