Related references
Note: Only part of the references are listed.Regulation of RAF Activity by 14-3-3 Proteins RAF KINASES ASSOCIATE FUNCTIONALLY WITH BOTH HOMO- AND HETERODIMERIC FORMS OF 14-3-3 PROTEINS
Andreas Fischer et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2009)
Bad Targets the Permeability Transition Pore Independent of Bax or Bak to Switch between Ca2+-Dependent Cell Survival and Death
Soumya Sinha Roy et al.
MOLECULAR CELL (2009)
C-RAF activation promotes BAD poly-ubiquitylation and turn-over by the proteasome
Jochen Fueller et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2008)
Isoform-specific interaction of C-RAF with mitochondria
Antoine Galmiche et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Positive regulation of A-RAF by phosphorylation of isoform-specific hinge segment and identification of novel phosphorylation sites
Angela Baljuls et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Phosphoproteome of resting human platelets
Rene P. Zahedi et al.
JOURNAL OF PROTEOME RESEARCH (2008)
The BCL-2 protein family: opposing activities that mediate cell death
Richard J. Youle et al.
NATURE REVIEWS MOLECULAR CELL BIOLOGY (2008)
Clostridium perfringens Delta Toxin Is Sequence Related to Beta Toxin, NetB, and Staphylococcus Pore-Forming Toxins, but Shows Functional Differences
Maria Manich et al.
PLOS ONE (2008)
Profiling phosphoproteins of yeast mitochondria reveals a role of phosphorylation in assembly of the ATP synthase
Joerg Reinders et al.
MOLECULAR & CELLULAR PROTEOMICS (2007)
Germline gain-of-function mutations in RAF1 cause Noonan syndrome
M. Abdur Razzaque et al.
NATURE GENETICS (2007)
Gain-of-function RAF1 mutations cause Noonan and LEOPARD syndromes with hypertrophic cardiomyopathy
Bhaswati Pandit et al.
NATURE GENETICS (2007)
Grb10 and active Raf-1 kinase promote bad-dependent cell survival
Sem Kebache et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
Apoptosis initiated when BH3 ligands engage multiple Bcl-2 homologs, not Bax or Bak
Simon N. Willis et al.
SCIENCE (2007)
Hierarchical regulation of mitochondrion-dependent apoptosis by BCL-2 subfamilies
Hyungjin Kim et al.
NATURE CELL BIOLOGY (2006)
How do Bax and Bak lead to permeabilization of the outer mitochondrial membrane?
Antonella Antignani et al.
CURRENT OPINION IN CELL BIOLOGY (2006)
Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?
L. M. Dejean et al.
CELL DEATH AND DIFFERENTIATION (2006)
Reversible membrane interaction of BAD requires two C-terminal lipid binding domains in conjunction with 14-3-3 protein binding
Mirko Hekman et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Bcl-2 changes conformation to inhibit Bax oligomerization
Paulina J. Dlugosz et al.
EMBO JOURNAL (2006)
被撤回的出版物: The Raf inhibitor BAY 43-9006 (Sorafenib) induces caspase-independent apoptosis in melanoma cells (Retracted article. See vol. 79, pg. 5459, 2019)
DJ Panka et al.
CANCER RESEARCH (2006)
Pim kinases phosphorylate multiple sites on Bad and promote 14-3-3 binding and dissociation from Bcl-XL
A Macdonald et al.
BMC CELL BIOLOGY (2006)
The BAD protein integrates survival signaling by EGFR/MAPK and PI3K/Akt kinase pathways in PTEN-deficient tumor cells
QB She et al.
CANCER CELL (2005)
BAD Ser128 is not phosphorylated by c-Jun NH2-terminal kinase for promoting apoptosis
JY Zhang et al.
CANCER RESEARCH (2005)
Bag1 is essential for differentiation and survival of hematopoietic and neuronal cells
R Götz et al.
NATURE NEUROSCIENCE (2005)
p21-Activated kinase 1 (Pak1)-dependent phosphorylation of Raf-1 regulates its mitochondrial localization, phosphorylation of BAD, and Bcl-2 association
SH Jin et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
A small molecule-kinase interaction map for clinical kinase inhibitors
MA Fabian et al.
NATURE BIOTECHNOLOGY (2005)
Exposure to hypoxia rapidly induces activity within a living synapse
EA Jonas et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function
L Chen et al.
MOLECULAR CELL (2005)
Dynamic changes in C-raf phosphorylation and 14-3-3 protein binding in response to growth factor stimulation - Differential roles of 14-3-3 protein binding sites
M Hekman et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
Bcl-2 proteins: master switches at the intersection of death signaling and the survival control by Raf kinases
UR Rapp et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH (2004)
Structural biology of the Bcl-2 family of proteins
AM Petros et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH (2004)
JNK suppresses apoptosis via phosphorylation of the proapoptotic Bcl-2 family protein BAD
CF Yu et al.
MOLECULAR CELL (2004)
Cell death: Critical control points
NN Danial et al.
CELL (2004)
Protein kinase CK2 phosphorylates BAD at threonine-117
S Klumpp et al.
NEUROCHEMISTRY INTERNATIONAL (2004)
Proapoptotic N-truncated BCL-xL protein activates endogenous mitochondrial channels in living synaptic terminals
EA Jonas et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2004)
The RAF proteins take centre stage
C Wellbrock et al.
NATURE REVIEWS MOLECULAR CELL BIOLOGY (2004)
BAY 43-9006 exhibits broad spectrum oral antitumor activity and targets the RAF/MEK/ERK pathway and receptor tyrosine kinases involved in tumor progression and angiogenesis
SM Wilhelm et al.
CANCER RESEARCH (2004)
Raf and the road to cell survival: a tale of bad spells, ring bearers and detours
J Troppmair et al.
BIOCHEMICAL PHARMACOLOGY (2003)
Protein kinase A blocks Raf-1 activity by stimulating 14-3-3 binding and blocking Raf-1 interaction with Ras
N Dumaz et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Spatial and temporal association of Bax with mitochondrial fission sites, Drp1, and Mfn2 during apoptosis
M Karbowski et al.
JOURNAL OF CELL BIOLOGY (2002)
Functional specificity in 14-3-3 isoform interactions through dimer formation and phosphorylation. Chromosome location of mammalian isoforms and variants.
A Aitken
PLANT MOLECULAR BIOLOGY (2002)
Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane
T Kuwana et al.
CELL (2002)
JNK phosphorylation and activation of BAD couples the stress-activated signaling pathway to the cell death machinery
N Donovan et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Mitochondrial respiratory control is lost during growth factor deprivation
E Gottlieb et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)
Associations of B- and C-raf with cholesterol, phosphatidylserine, and lipid second messengers - Preferential binding of Raf to artificial lipid rafts
M Hekman et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Activation of JNK1, RSK2, and MSK1 is involved in serine 112 phosphorylation of bad by ultraviolet B radiation
QB She et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Negative regulation of mitochondrial VDAC channels by C-Raf kinase
V Le Mellay et al.
BMC CELL BIOLOGY (2002)
Cdc2 phosphorylation of BAD links the cell cycle to the cell death machinery
Y Konishi et al.
MOLECULAR CELL (2002)
Identification of a novel phosphorylation site, Ser-170, as a regulator of bad pro-apoptotic activity
S Dramsi et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
The structure of the C-terminal domain of the pro-apoptotic protein bak and its interaction with model membranes
MD Martinez-Senac et al.
BIOPHYSICAL JOURNAL (2002)
The role of dynamin-related protein 1, a mediator of mitochondrial fission, in apoptosis
S Frank et al.
DEVELOPMENTAL CELL (2001)
The serine/threonine kinase PAK4 prevents caspase activation and protects cells from apoptosis
N Gnesutta et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
Solution structure of the antiapoptotic protein bcl-2
AM Petros et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2001)
Insights into programmed cell death through structural biology
SW Fesik
CELL (2000)
Phosphorylation of the pro-apoptotic protein BAD on serine 155, a novel site, contributes to cell survival
K Virdee et al.
CURRENT BIOLOGY (2000)
Study of the secondary structure of the C-terminal domain of the antiapoptotic protein Bcl-2 and its interaction with model membranes
MD Martínez-Senac et al.
BIOCHEMISTRY (2000)
14-3-3 proteins and survival kinases cooperate to inactivate BAD by BH3 domain phosphorylation
SR Datta et al.
MOLECULAR CELL (2000)
Rsk1 mediates a MEK-MAP kinase cell survival signal
A Shimamura et al.
CURRENT BIOLOGY (2000)
The Akt proto-oncogene links Ras to Pak and cell survival signals
Y Tang et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
p21-activated kinase 1 phosphorylates the death agonist Bad and protects cells from apoptosis
A Schurmann et al.
MOLECULAR AND CELLULAR BIOLOGY (2000)
Share Paper
