Related references
Note: Only part of the references are listed.Follow-up Study from Birth of the Effects of Prenatal Stresses
D. H. Stott
DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY (2010)
Effects of cyclooxygenase-2 gene inactivation on cardiac autonomic and left ventricular function in experimental diabetes
Aaron P. Kellogg et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2009)
Diverse clinical spectrum of stress-induced cardiomyopathy
Yian-Ping Lee et al.
INTERNATIONAL JOURNAL OF CARDIOLOGY (2009)
Oxidative stress and adenosine A(1) receptor activation differentially modulate subcellular cardiomyocyte MAPKs
Cherry Ballard-Croft et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2008)
Important role of p38 MAP kinase/NF-κB signaling pathway in the sepsis-induced conversion of cardiac myocytes to a proinflammatory phenotype
Min Yang et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2008)
p38 MAPK mediates COX-2 gene expression by corticosterone in cardiomyocytes
Haipeng Sun et al.
CELLULAR SIGNALLING (2008)
Prenatal stress does not alter innate novelty-seeking behavioral traits, but differentially affects individual differences in neuroendocrine stress responsivity
Sarah Clinton et al.
PSYCHONEUROENDOCRINOLOGY (2008)
Inducible nitric oxide synthase attenuates adrenergic signaling in alcohol fed rats
Deepak Khanna et al.
JOURNAL OF CARDIOVASCULAR PHARMACOLOGY (2007)
Influence of mental stress on cardiovascular function as evaluated by changes in energy expenditure
Asuka Sawai et al.
HYPERTENSION RESEARCH (2007)
Acute β-adrenergic overload produces myocyte damage through calcium leakage from the ryanodine receptor 2 but spares cardiac stem cells
Georgina M. Ellison et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
p38-MAPK induced dephosphorylation of α-tropomyosin is associated with depression of myocardial sarcomeric tension and ATPase activity
Susan Vahebi et al.
CIRCULATION RESEARCH (2007)
Changes in plasma volume associated with mental stress ischemia in patients with coronary artery disease
Simon L. Bacon et al.
INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY (2006)
iPLA2 inhibitor blocks negative inotropic effect of HIV gp120 on cardiac myocytes
H Kan et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2006)
Neurohumoral features of myocardial stunning due to sudden emotional stress
IS Wittstein et al.
NEW ENGLAND JOURNAL OF MEDICINE (2005)
Acute and reversible cardiomyopathy provoked by stress in women from the United States
SW Sharkey et al.
CIRCULATION (2005)
p38 MAP kinase inhibitor reverses stress-induced cardiac myocyte dysfunction
H Kan et al.
JOURNAL OF APPLIED PHYSIOLOGY (2005)
Acute p38 MAPK activation decreases force development in ventricular myocytes
Y Chen et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2003)
Inflammatory mediators and reversible myocardial dysfunction
H Kan et al.
JOURNAL OF CELLULAR PHYSIOLOGY (2003)
Mitogen-activated protein kinase pathways mediated by ERK, JNK, and p38 protein kinases
GL Johnson et al.
SCIENCE (2002)
In the cellular garden of forking paths: How p38 MAPKs signal for downstream assistance
Y Shi et al.
BIOLOGICAL CHEMISTRY (2002)
p38 mitogen-activated protein kinase mediates a negative inotropic effect in cardiac myocytes
P Liao et al.
CIRCULATION RESEARCH (2002)
Tako-tsubo-like left ventricular dysfunction with ST-segment elevation: A novel cardiac syndrome mimicking acute myocardial infarction
S Kurisu et al.
AMERICAN HEART JOURNAL (2002)
The in vivo role of p38 MAP kinases in cardiac remodeling and restrictive cardiomyopathy
P Liao et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2001)
β2-adrenergic receptor-induced p38 MAPK activation is mediated by protein kinase A rather than by Gi or Gβγ in adult mouse cardiomyocytes
M Zheng et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
Effects of prenatal stress on defensive withdrawal behavior and corticotropin releasing factor systems in rat brain
HE Ward et al.
PHYSIOLOGY & BEHAVIOR (2000)
SB 203580, a mitogen-activated protein kinase inhibitor, abolishes resistance to myocardial infarction induced by heat stress
M Joyeux et al.
CARDIOVASCULAR DRUGS AND THERAPY (2000)