Journal
MICROBES AND ENVIRONMENTS
Volume 30, Issue 3, Pages 284-287Publisher
JAPANESE SOC MICROBIAL ECOLOGY, DEPT BIORESOURCE SCIENCE
DOI: 10.1264/jsme2.ME15046
Keywords
Porcine epidemic diarrhea; microbiota; 16S rRNA gene; large intestine; next-generation sequencing
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Funding
- Business for Cooperative R&D between Industry, Academy, and Research Institute funded Korea Small and Medium Business Administration [C0250398]
- Ministry for Food, Agriculture, Forestry and Fisheries, Republic of Korea [112013032SB010]
- Institute of Planning & Evaluation for Technology in Food, Agriculture, Forestry & Fisheries (iPET), Republic of Korea [112013032SB010, 112013033SB010] Funding Source: Korea Institute of Science & Technology Information (KISTI), National Science & Technology Information Service (NTIS)
- Korea Evaluation Institute of Industrial Technology (KEIT) [C0250398] Funding Source: Korea Institute of Science & Technology Information (KISTI), National Science & Technology Information Service (NTIS)
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The gastrointestinal tract of mammals is a complex ecosystem with distinct environments and comprises hundreds of different types of bacterial cells. The gut microbiota may play a critical role in the gut health of the host. We herein attempted to identify a microbiota shift that may be affected by porcine epidemic diarrhea (PED). We observed significant differences in microbiota between the control and PED virus (PEDV)-infected groups at both the phylum and genus level. Most commensal bacteria (i.e. Psychrobacter, Prevotella, and Faecalibacterium) in the healthy gastrointestinal tract were decreased due to dysbiosis induced by PEDV infection.
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